Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

<i>MGA</i>, a suppressor of<i>MYC</i>, is recurrently inactivated in high risk chronic lymphocytic leukemia

5 Oct 2012

Abstract excerpt

Although modern treatment strategies are highly effective in many cases of progressive chronic lymphocytic leukemia (CLL), a challenging subgroup of patients show poor response to standard regimens...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer-related Molecular Pathways
Share to Chronic Lymphocytic Leukemia Research
Share to Lymphoma Diagnosis and Treatment

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 23039309
DOI: 10.3109/10428194.2012.723706

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

<i>MGA</i>, a suppressor of<i>MYC</i>, is recurrently inactivated in high risk chronic lymphocytic leukemiaPMID 23039309Read ↗

ZMYM3 mutations modulate histone acetylation and cooperate with NOTCH1 mutations in chronic lymphocytic leukemiaSemantic proximity Read ↗

<i>SAMHD1</i> mutations in mantle cell lymphoma are recurrent and confer <i>in vitro</i> resistance to nucleoside analoguesSemantic proximity Read ↗

New drug discovery approaches targeting recurrent mutations in chronic lymphocytic leukemia.Semantic proximity Read ↗

Modeling <i>IKZF1</i> lesions in B-ALL reveals distinct chemosensitivity patterns and potential therapeutic vulnerabilitiesSemantic proximity Read ↗

<i>RAS</i> mutations drive proliferative chronic myelomonocytic leukemia via activation of a novel KMT2A-PLK1 axisSemantic proximity Read ↗

ZMYM3 mutations modulate histone acetylation and cooperate with NOTCH1 mutations in chronic lymphocytic leukemia.Semantic proximity Read ↗

Meis1 is critical to the maintenance of human acute myeloid leukemia cells independent of MLL rearrangementsSemantic proximity Read ↗

KAT7 is a therapeutic vulnerability of <i>MLL</i> -rearranged acute myeloid leukemiaSemantic proximity Read ↗

Functional Characterization of Cooperating MGA Mutations in RUNX1::RUNX1T1 Acute Myeloid LeukemiaSemantic proximity Read ↗

Efficacy of Ruxolitinib in Chronic Eosinophilic Leukemia Associated With a <i>PCM1-JAK2</i> Fusion GeneSemantic proximity Read ↗

The non-genomic loss of function of tumor suppressors: an essential role in the pathogenesis of chronic myeloid leukemia chronic phase.Semantic proximity Read ↗

Selective Enhancer Dependencies in <i>MYC</i> -Intact and <i>MYC</i> -Rearranged Germinal Center B-cell Diffuse Large B-cell LymphomaSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.