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Article

Escherichia coli strains with multiple DNA repair defects are hyperinduced for the SOS response.

Journal of bacteriology - 1 Aug 1990

Foster P L

Abstract excerpt

Escherichia coli strains defective for the repair of apurinic/apyrimidinic sites and for the UvrABC excision repair pathway could be constructed if they also carried a mutation in ung, which encodes uracil glycosylase, or sulA, which encodes an SOS-inducible inhibitor of septation. The resultant strains were sensitive to alkylation damage and hyperinduced for the SOS response, but had unpredictable spontaneous...

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Identifiers

PMID: 2115878
DOI: 10.1128/jb.172.8.4719-4720.1990
PMCID: PMC213313

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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Escherichia coli strains with multiple DNA repair defects are hyperinduced for the SOS response.PMID 2115878Read ↗

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The effect of hyper-induced SOS response on evolution in Escherichia coli.Semantic proximity Read ↗

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Atypical Role for PhoU in Mutagenic Break Repair under Stress in Escherichia coli.Semantic proximity Read ↗

SOS and UVM pathways have lesion-specific additive and competing effects on mutation fixation at replication-blocking DNA lesions.Semantic proximity Read ↗

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The SOS response regulates adaptive mutation.Semantic proximity Read ↗

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DNA alkylation repair limits spontaneous base substitution mutations in Escherichia coli.Semantic proximity Read ↗

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