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Article

Telomere instability in a human tumor cell line expressing a dominant-negative WRN protein

1 Sept 2003

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Share to DNA Repair Mechanisms
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Share to Telomeres, Telomerase, and Senescence

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 12827497
DOI: 10.1007/s00439-003-0972-y

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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Telomere instability in a human tumor cell line expressing a dominant-negative WRN proteinPMID 12827497Read ↗

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Telomere dysfunction as a cause of genomic instability in Werner syndromeSemantic proximity Read ↗

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Functional role of the Werner syndrome RecQ helicase in human fibroblasts.Semantic proximity Read ↗

Response to Replication Stress and Maintenance of Genome Stability by WRN, the Werner Syndrome ProteinSemantic proximity Read ↗

Loss of p21 promoted tumorigenesis in the background of telomere dysfunctions induced by TRF2 and Wrn deficiency.Semantic proximity Read ↗

WRN as a Novel Target of Synthetic Lethality: Current Advances and Future PerspectivesSemantic proximity Read ↗

Role of Werner syndrome gene product helicase in carcinogenesis and in resistance to genotoxins by cancer cells.Semantic proximity Read ↗

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