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Differential adenosine to inosine RNA editing of SINE B2 non-coding RNAs in mouse unveils a novel type of epi-transcriptome response to amyloid beta neuro-toxicity

2026-01-31

Abstract excerpt

<h4>ABSTRACT</h4> Alzheimer’s disease (AD) is characterized by early molecular responses to amyloid beta neuro-toxicity that remain poorly defined. RNA editing by adenosine-to-inosine (A-to-I) conversion is a major epitranscriptomic mechanism, yet its contribution to non-coding RNA regulation during neurodegeneration is largely unknown. Short Interspersed Nuclear Element (SINE)–derived RNAs, including mouse B2 RN...

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Literature Corpus work
3934322a-db95-55f9-9aef-46271a4076bb
DOI
10.64898/2026.01.30.702824
Open publication

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Differential adenosine to inosine RNA editing of SINE B2 non-coding RNAs in mouse unveils a novel type of epi-transcriptome response to amyloid beta neuro-toxicityDOI 10.64898/2026.01.30.702824
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