Hepatitis B Virus Infection Among Leprosy Patients: A Case for Polymorphisms Compromising Activation of the Lectin Pathway and Complement Receptors.

doi:10.3389/fimmu.2020.574457

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Hepatitis B Virus Infection Among Leprosy Patients: A Case for Polymorphisms Compromising Activation of the Lectin Pathway and Complement Receptors.

Frontiers in immunology - 1 Jan 2020

Boldt Angelica Beate Winter, Oliveira-Toré Camila de Freitas, Kretzschmar Gabriela Canalli, Weinschutz Mendes Hellen, Stinghen Sérvio Túlio, Andrade Fabiana Antunes, Bumiller-Bini Valéria, Gonçalves Letícia Boslooper, Braga Anna Carolina de Moraes, Stahlke Ewalda von Rosen Seeling, Velavan Thirumalaisamy P, Thiel Steffen, de Messias-Reason Iara José Taborda

Abstract excerpt

Thousands of leprosy patients not only suffer from physical deformities, but also either have or have had hepatitis B virus (HBV) coinfection. Polymorphisms of the complement system modulate susceptibility to leprosy, but genetic susceptibility to past or present HBV infection is unknown. We used sequencing and multiplex sequence-specific PCR to genotype 72 polymorphisms of seven genes (MBL2, FCN1, FCN2, FCN3,...

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Identifiers

PMID: 33643280
DOI: 10.3389/fimmu.2020.574457
PMCID: PMC7904891

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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