Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Transcriptomic profiling of chemical exposure reveals roles of Yap1 in protecting yeast cells from oxidative and other types of stresses.

Yeast (Chichester, England) - 1 Jan 2016

Zhang Chao, Li Zhouquan, Zhang Xiaohua, Yuan Li, Dai Heping, Xiao Wei

Abstract excerpt

Transcriptomic profiles are generated by comparing wild-type and the yeast yap1 mutant to various chemicals in an attempt to establish a correlation between this gene mutation and chemical exposure. Test chemicals include ClonNAT as a non-genotoxic agent, methyl methanesulphonate (MMS) as an alkylating agent, tert-butyl hydroperoxide (t-BHP) as an oxidative agent and the mixture of t-BHP and MMS to reflect...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to beta-Galactosidase
Share to Ion Transport

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Metabolic Networks and Pathways

Share to Oxidation-Reduction

Share to Oxidative Stress

Share to Peroxisomes

Share to Toxicogenetics

Share to Transcriptional Activation

Source

Identifiers

PMID: 26389527
DOI: 10.1002/yea.3135

Genes

YAP1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Transcriptomic profiling of chemical exposure reveals roles of Yap1 in protecting yeast cells from oxidative and other types of stresses.PMID 26389527Read ↗

High-throughput phenotypic profiling of gene-environment interactions by quantitative growth curve analysis in Saccharomyces cerevisiae.Semantic proximity Read ↗

Genomic phenotyping of the essential and non-essential yeast genome detects novel pathways for alkylation resistance.Semantic proximity Read ↗

Toxicogenomics and systems toxicology: aims and prospects.Semantic proximity Read ↗

The adaptive response of anaerobically grown Saccharomyces cerevisiae to hydrogen peroxide is mediated by the Yap1 and Skn7 transcription factors.Semantic proximity Read ↗

Yap1: a DNA damage responder in Saccharomyces cerevisiae.Semantic proximity Read ↗

Integrating phenotypic and expression profiles to map arsenic-response networks.Semantic proximity Read ↗

Activity of the Yap1 transcription factor in Saccharomyces cerevisiae is modulated by methylglyoxal, a metabolite derived from glycolysis.Semantic proximity Read ↗

Knowledge-Driven Approaches to Create the MTox700+ Metabolite Panel for Predicting Toxicity.Semantic proximity Read ↗

Unbiased Approach for the Identification of Molecular Mechanisms Sensitive to Chemical ExposuresSemantic proximity Read ↗

Damage recovery pathways in Saccharomyces cerevisiae revealed by genomic phenotyping and interactome mapping.Semantic proximity Read ↗

Yippee-like protein Moh1 links gene expression to metabolism and selective stress resistance in <i>Saccharomyces cerevisiae</i>Semantic proximity Read ↗

Genomic phenotyping by barcode sequencing broadly distinguishes between alkylating agents, oxidizing agents, and non-genotoxic agents, and reveals a role for aromatic amino acids in cellular recovery after quinone exposure.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.