Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Neointimal hyperplasia and vasoreactivity are controlled by genetic elements on rat chromosome 3.

Hypertension (Dallas, Tex. : 1979) - 1 Feb 2010

Nestor Kalinoski Andrea L, Ramdath Ramona S, Langenderfer Kay M, Sikanderkhel Saad, Deraedt Sarah, Welch Marlene, Park James L, Pringle Timothy, Joe Bina, Cicila George T, Allison David C

Abstract excerpt

Neointimal hyperplasia (NIH) can lead to restenosis after clinical vascular interventions. NIH results from complex and poorly understood interactions between signaling cascades in the extracellular matrix and the disrupted endothelium, which lead to vessel occlusion. Quantitative trait loci (QTLs) were reported previously on rat chromosomes 3 and 6 through linkage analysis of postinjury NIH in midiliac arterial...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Animals, Congenic
Share to Chromosomes, Human, Pair 3

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Constriction, Pathologic

Share to Femoral Artery

Share to Hyperplasia

Share to Rats, Inbred BN

Share to Rats, Inbred SHR

Share to Tunica Intima

Source

Open in PubMed Open full text

Identifiers

PMID: 20026763
DOI: 10.1161/HYPERTENSIONAHA.109.142505
PMCID: PMC3765071

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Neointimal hyperplasia and vasoreactivity are controlled by genetic elements on rat chromosome 3.PMID 20026763Read ↗

RNO3 QTL regulates vascular structure and arterial stiffness in the spontaneously hypertensive rat.Semantic proximity Read ↗

Quantitative trait locus analysis of neointimal formation in an intercross between C57BL/6 and C3H/HeJ apolipoprotein E-deficient mice.Semantic proximity Read ↗

Genetic architecture underlying variation in extent and remodeling of the collateral circulation.Semantic proximity Read ↗

Genetic loci that control the angiogenic response to basic fibroblast growth factor.Semantic proximity Read ↗

Identification of two susceptibility loci for vascular fragility in the Brown Norway rat.Semantic proximity Read ↗

Identification of a region of rat chromosome 1 that impairs the myogenic response and autoregulation of cerebral blood flow in fawn-hooded hypertensive rats.Semantic proximity Read ↗

Fetal origins of adult vascular dysfunction in mice lacking endothelial nitric oxide synthaseSemantic proximity Read ↗

Synergistic QTL interactions between Rf-1 and Rf-3 increase renal damage susceptibility in double congenic rats.Semantic proximity Read ↗

Transgenerational effect of fetal programming on vascular phenotype and reactivity in endothelial nitric oxide synthase knockout mouse model.Semantic proximity Read ↗

Spontaneous elastic tissue lesions in the rat abdominal aorta, a genetically determined phenotype.Semantic proximity Read ↗

Multiple blood pressure loci on rat chromosome 13 attenuate development of hypertension in the Dahl S hypertensive rat.Semantic proximity Read ↗

Knockout of the NONO Gene Inhibits Neointima Formation in a Mouse Model of Vascular Injury.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.