NFY or a related CCAAT binding factor can be replaced by other transcriptional activators for co-operation with HNF1 in driving the rat albumin promoter in vivo.

doi:10.1016/0022-2836(91)90735-o

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Article

NFY or a related CCAAT binding factor can be replaced by other transcriptional activators for co-operation with HNF1 in driving the rat albumin promoter in vivo.

Journal of molecular biology - 5 Nov 1991

Tronche F, Rollier A, Sourdive D, Cereghini S, Yaniv M

Abstract excerpt

Like many eukaryotic genes, the rat albumin promoter contains a CCAAT consensus motif at position -80. In transfected H4II hepatoma cells the strength of this promoter depends to a large extent on the integrity of a hepatic nuclear factor 1 (HNF1) binding site located at position -60 and to a les...

Topics

Adenoviridae
Albumins
Animals
Base Sequence
CCAAT-Enhancer-Binding Proteins
DNA
DNA-Binding Proteins
Fibrinogen
Fructose-Bisphosphate Aldolase
Hepatocyte Nuclear Factor 1
Hepatocyte Nuclear Factor 1-alpha
Hepatocyte Nuclear Factor 1-beta
Liver Neoplasms, Experimental
Molecular Sequence Data
Mutation
Nuclear Proteins
Promoter Regions, Genetic
Rats

Source

Open in PubMed

Identifiers

PMID: 1942067
DOI: 10.1016/0022-2836(91)90735-o

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 30 Aug 2026. Prepared record publication-1942067-2026-08-30.1. Manifest publication-graph-cohort-2026-08-30.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 30 Aug 2026.

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