Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Absence of TP53 codon 249 mutations in young Guinean children with high aflatoxin exposure.

Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology - 1 Aug 2005

Turner Paul C, Sylla Abdoulaye, Kuang Shuang-Yuan, Marchant Clare L, Diallo Mamadou S, Hall Andrew J, Groopman John D, Wild Christopher P

Abstract excerpt

Infection with hepatitis viruses and chronic exposure to high levels of dietary aflatoxins are the major etiologic agents for hepatocellular carcinoma in west Africa. A challenge for the prevention of hepatocellular carcinoma in this region is that both hepatitis B virus and aflatoxin exposures start early in life; indeed, aflatoxin exposures can start in utero and continue unabated throughout childhood. A...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adenoma, Liver Cell
Share to Adolescent

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Aflatoxins

Share to Child, Preschool

Share to DNA, Neoplasm

Share to Genes, p53

Share to Liver Neoplasms

Source

Identifiers

PMID: 16103461
DOI: 10.1158/1055-9965.EPI-04-0923

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Absence of TP53 codon 249 mutations in young Guinean children with high aflatoxin exposure.PMID 16103461Read ↗

249(ser) TP53 mutation in plasma DNA, hepatitis B viral infection, and risk of hepatocellular carcinoma.Semantic proximity Read ↗

The aflatoxin-induced TP53 mutation at codon 249 (R249S): biomarker of exposure, early detection and target for therapy.Semantic proximity Read ↗

Ser-249TP53 mutation in tumour and plasma DNA of hepatocellular carcinoma patients from a high incidence area in the Gambia, West Africa.Semantic proximity Read ↗

TP53 R249S mutations, exposure to aflatoxin, and occurrence of hepatocellular carcinoma in a cohort of chronic hepatitis B virus carriers from Qidong, China.Semantic proximity Read ↗

p53 mutation in hepatocellular carcinoma after aflatoxin exposure.Semantic proximity Read ↗

249ser p53 mutation in the serum of black southern African patients with hepatocellular carcinoma.Semantic proximity Read ↗

Ser-249 p53 mutations in plasma DNA of patients with hepatocellular carcinoma from The Gambia.Semantic proximity Read ↗

Analysis of the p53 tumor-suppressor gene in hepatocellular carcinomas from Britain.Semantic proximity Read ↗

Seasonal variation in TP53 R249S-mutated serum DNA with aflatoxin exposure and hepatitis B virus infection.Semantic proximity Read ↗

High prevalence of mutations at codon 249 of the p53 gene in hepatocellular carcinomas from Senegal.Semantic proximity Read ↗

Quantitative analysis of plasma TP53 249Ser-mutated DNA by electrospray ionization mass spectrometry.Semantic proximity Read ↗

Hepatocellular carcinoma: from gene to public health.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.