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The IL-33/ST2 Axis Promotes Post-COVID-19 Pulmonary Fibrosis Through β-catenin/PPAR-γ-mediated Epithelial-Mesenchymal Transition

2025-09-09

Abstract excerpt

<title>Abstract</title> <p>Background Post-COVID-19 pulmonary fibrosis (PCPF) presents an increasingly significant public health challenge. Although fibrotic mechanisms in PCPF are extensively studied, their exact pathogenesis remains unresolved. Interleukin-33 (IL-33), a critical alarmin in viral infections, including COVID-19, has been reported to drive fibrotic processes across multiple organs, yet its role in...

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Literature Corpus work
bf89d1ac-858f-5866-97d6-7c25567cea09
DOI
10.21203/rs.3.rs-7024312/v1
Open publication

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The IL-33/ST2 Axis Promotes Post-COVID-19 Pulmonary Fibrosis Through β-catenin/PPAR-γ-mediated Epithelial-Mesenchymal TransitionDOI 10.21203/rs.3.rs-7024312/v1
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