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Flightless I Homolog Reverses Neo-Endocrine Therapy Resistance through YBX1/PD-L1 Axis Mediated-Immune Evasion in Prostate Cancer

2020-08-01

Abstract excerpt

<h4>Background: </h4> Tumor cells can evade immune surveillance and immune killing effects during neo-endocrine therapy resistance, but the mechanisms that induce this phenomenon are still unclear. Flightless I homolog (FLII) functions as a coregulator of transcription factors in malignancies. Here, we aim to identify the functions and mechanisms of FLII-mediated neo-endocrine therapy resistance and immune evasion...

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Literature Corpus work
4bb76b88-88ce-5301-bfc7-125d6a1ba45b
DOI
10.21203/rs.3.rs-48777/v1
Open publication

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Flightless I Homolog Reverses Neo-Endocrine Therapy Resistance through YBX1/PD-L1 Axis Mediated-Immune Evasion in Prostate CancerDOI 10.21203/rs.3.rs-48777/v1
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