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Article

AT1 receptor A/C1166 polymorphism contributes to cardiac hypertrophy in subjects with hypertrophic cardiomyopathy.

Hypertension (Dallas, Tex. : 1979) - 1 Nov 1998

Osterop A P, Kofflard M J, Sandkuijl L A, ten Cate F J, Krams R, Schalekamp M A, Danser A H

Abstract excerpt

The development of left ventricular hypertrophy (LVH) in subjects with hypertrophic cardiomyopathy (HCM) is variable, suggesting a role for modifying factors such as angiotensin II. We investigated whether the angiotensin II type 1 receptor (AT1-R) A/C1166 polymorphism, the angiotensin-converting enzyme (ACE) insertion/deletion (I/D) polymorphism, and/or plasma renin influence LVH in HCM. Left ventricular mass...

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Identifiers

PMID: 9822439
DOI: 10.1161/01.hyp.32.5.825

Genes

ACE

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

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AT1 receptor A/C1166 polymorphism contributes to cardiac hypertrophy in subjects with hypertrophic cardiomyopathy.PMID 9822439Read ↗

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Angiotensin-I converting enzyme genotypes and left ventricular hypertrophy in patients with hypertrophic cardiomyopathy.Semantic proximity Read ↗

Role of angiotensin II type I (AT1 A1166C) receptor polymorphism in susceptibility of left ventricular dysfunction.Semantic proximity Read ↗

Functional polymorphisms in genes of the Angiotensin and Serotonin systems and risk of hypertrophic cardiomyopathy: AT1R as a potential modifier.Semantic proximity Read ↗

Renal function dependent association of AGTR1 polymorphism (A1166C) and electrocardiographic left-ventricular hypertrophy.Semantic proximity Read ↗

alpha-Adducin Gly460Trp polymorphism, left ventricular mass and plasma renin activity.Semantic proximity Read ↗

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