Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

3'UTR polymorphism of Thymidylate Synthase gene increased the risk of persistence of pre-neoplastic cervical lesions.

BMC cancer - 15 Apr 2020

Silva Nayara Nascimento Toledo, Santos Ana Carolina Silva, Nogueira Verlândia Mendes, Carneiro Cláudia Martins, Lima Angélica Alves

Abstract excerpt

BACKGROUND: Cervical cancer is caused by high-risk Human Papillomavirus (hr-HPV) infection associated with cofactors that has been analyzed as predictors of the remission or persistence of cytological abnormalities remission or persistence. These cofactors can be either environmental, epigenetic, or genetic. Polymorphism in genes of enzymes that act on one-carbon metabolism alter their activity and also may be...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to 3' Untranslated Regions
Share to 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to DNA, Viral

Share to Papillomaviridae

Share to Papillomavirus Infections

Share to Precancerous Conditions

Share to Thymidylate Synthase

Share to Uterine Cervical Neoplasms

Source

Open in PubMed Open full text

Identifiers

PMID: 32295543
DOI: 10.1186/s12885-020-06811-7
PMCID: PMC7161242

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

3'UTR polymorphism of Thymidylate Synthase gene increased the risk of persistence of pre-neoplastic cervical lesions.PMID 32295543Read ↗

3’UTR Polymorphism of Thymidylate Synthase gene increased the risk of persistence of pre-neoplastic cervical lesionsSemantic proximity Read ↗

Genetic polymorphisms in one-carbon metabolism increased the risk of persistence of pre-neoplastic cervical lesionsSemantic proximity Read ↗

Methylenetetrahydrofolate reductase (MTHFR) polymorphisms and promoter methylation in cervical oncogenic lesions and cancer.Semantic proximity Read ↗

The effect of methylenetetrahydrofolate reductase polymorphism C677T on cervical cancer in Korean women.Semantic proximity Read ↗

Common genetic variants and risk for HPV persistence and progression to cervical cancer.Semantic proximity Read ↗

Persistence of type-specific human papillomavirus infection and increased long-term risk of cervical cancer.Semantic proximity Read ↗

The effects of polymorphisms in methylenetetrahydrofolate reductase (MTHFR), methionine synthase (MTR), and methionine synthase reductase (MTRR) on the risk of cervical intraepithelial neoplasia and cervical cancer in Korean women.Semantic proximity Read ↗

Cervical cancer pathogenesis is associated with one-carbon metabolism.Semantic proximity Read ↗

Folate deficiency modifies the risk of CIN3+ associated with DNA methylation levels: a nested case–control study from the ASCUS-COL trialSemantic proximity Read ↗

Association of glutathione S-transferase M1 and T1 null polymorphisms with the development of cervical lesions: a meta-analysis.Semantic proximity Read ↗

Development of cervical intraepithelial lesions and cervical cancer is not influenced by SOD2 RS4880 polymorhism.Semantic proximity Read ↗

Single nucleotide polymorphisms in the PRDX3 and RPS19 and risk of HPV persistence and cervical precancer/cancer.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.