Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Genetic Depletion or Hyperresponsiveness of Natural Killer Cells Do Not Affect Atherosclerosis Development

18 Oct 2017

Abstract excerpt

Rationale: Chronic inflammation is central in the development of atherosclerosis. Both innate and adaptive immunities are involved. Although several studies have evaluated the functions of natural killer (NK) cells in experimental animal models of atherosclerosis, it is not yet clear whether NK cells behave as protective or proatherogenic effectors. One of the main caveats of previous studies was the lack of...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adipokines, Inflammation, and Metabolic Diseases
Share to Atherosclerosis and Cardiovascular Diseases
Share to Immune Cell Function and Interaction

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 29046274
DOI: 10.1161/circresaha.117.311743

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Genetic Depletion or Hyperresponsiveness of Natural Killer Cells Do Not Affect Atherosclerosis DevelopmentPMID 29046274Read ↗

Quantitative and qualitative differences in proatherogenic NKT cells in apolipoprotein E-deficient mice.Semantic proximity Read ↗

<i>Apolipoprotein E</i> reduces the number and activation of non-invariant Natural Killer T cellsSemantic proximity Read ↗

Combined B, T and NK Cell Deficiency Accelerates Atherosclerosis in BALB/c Mice.Semantic proximity Read ↗

Development of spontaneous anergy in invariant natural killer T cells in a mouse model of dyslipidemia.Semantic proximity Read ↗

Natural killer T cells in lipoprotein metabolism and atherosclerosis.Semantic proximity Read ↗

Expression of the Lyst(beige) mutation is atheroprotective in chow-fed apolipoprotein E-deficient mice.Semantic proximity Read ↗

Prevention of oxLDL uptake leads to decreased atherosclerosis in hematopoietic NPC1-deficient Ldlr-/- mice.Semantic proximity Read ↗

Effect of natural killer T cell activation on initiation of atherosclerosisSemantic proximity Read ↗

T helper-cell phenotype regulates atherosclerosis in mice under conditions of mild hypercholesterolemia.Semantic proximity Read ↗

Expression, Function, and Molecular Properties of the Killer Receptor Ncr1-Noé.Semantic proximity Read ↗

Bone marrow-specific knock-in of a non-activatable Ikkα kinase mutant influences haematopoiesis but not atherosclerosis in Apoe-deficient mice.Semantic proximity Read ↗

HMG-CoA reductase promotes protein prenylation and therefore is indispensible for T-cell survival.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.