Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Expression of the Lyst(beige) mutation is atheroprotective in chow-fed apolipoprotein E-deficient mice.

Journal of lipid research - 1 Feb 2008

Petrovan Ramona J, Yuan Yuan, Curtiss Linda K

Abstract excerpt

Lyst(beige) mice crossed with hyperlipidemic low density lipoprotein receptor-deficient mice (BgLDLr(-/-)) display increased lesion area and a more stable lesion morphology. To verify that the beige phenotype is not unique to LDLr(-/-) mice, we examined atherosclerosis in beige, apolipoprotein E-...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Animal Feed
Share to Apolipoproteins E
Share to Atherosclerosis

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Dietary Fats

Share to Intracellular Signaling Peptides and Proteins

Share to Proteins

Share to Receptors, LDL

Share to Vesicular Transport Proteins

Source

Identifiers

PMID: 17982137
DOI: 10.1194/jlr.M700410-JLR200

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-17982137-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Expression of the Lyst(beige) mutation is atheroprotective in chow-fed apolipoprotein E-deficient mice.PMID 17982137Read ↗

Alternate models of acute dyslipidemia reveal divergent pathways upon atherosclerosis initiationSemantic proximity Read ↗

CalDAG-GEFI Deficiency Reduces Atherosclerotic Lesion Development in Mice.Semantic proximity Read ↗

GSDME-mediated pyroptosis promotes the progression and associated inflammation of atherosclerosisSemantic proximity Read ↗

Spontaneous Hypercholesterolemia and Arterial Lesions in Mice Lacking Apolipoprotein ESemantic proximity Read ↗

HDL deficiency in genetically engineered mice requires elevated LDL to accelerate atherogenesis.Semantic proximity Read ↗

DBA/2J Haplotype on Distal Chromosome 2 Reduces Mertk Expression, Restricts Efferocytosis, and Increases Susceptibility to Atherosclerosis.Semantic proximity Read ↗

Effects of hyperfibrinogenemia on vasculature of C57BL/6 mice with and without atherogenic diet.Semantic proximity Read ↗

Do the Apoe-/- and Ldlr-/- Mice Yield the Same Insight on Atherogenesis?Semantic proximity Read ↗

Hyperlipidemia induces typical atherosclerosis development in Ldlr and Apoe deficient rats.Semantic proximity Read ↗

Macrophage-Specific Expression of Human Lipoprotein Lipase Accelerates Atherosclerosis in Transgenic Apolipoprotein E Knockout Mice but Not in C57BL/6 MiceSemantic proximity Read ↗

Macrophage apolipoprotein E reduces atherosclerosis and prevents premature death in apolipoprotein E and scavenger receptor-class BI double-knockout mice.Semantic proximity Read ↗

The emergence of mouse models of atherosclerosis and their relevance to clinical researchSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.