Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Increased expression of NTPDases 2 and 3 in mesenteric endothelial cells during schistosomiasis favors leukocyte adhesion through P2Y1 receptors.

Vascular pharmacology - 1 Jul 2016

Oliveira Suellen Darc Santos, Oliveira Nathália F, Meyer-Fernandes José R, Savio Luiz Eduardo Baggio, Ornelas Flavia G I, Ferreira Zulma S, Coutinho-Silva Robson, Silva Claudia Lucia Martins

Abstract excerpt

Schistosomiasis is caused by an intravascular parasite and linked to phenotypic changes in endothelial cells that favor inflammation. Endothelial cells express P2Y1 receptors (P2Y1R), and their activation by ADP favors leukocyte adhesion to the endothelial monolayer. We aimed to evaluate the influence of schistosomiasis upon endothelial purinergic signaling-mediated leukocyte adhesion. Mesenteric endothelial...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adenosine Diphosphate
Share to Coculture Techniques

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Endothelial Cells

Share to Hydrolysis

Share to Leukocytes

Share to Mesentery

Share to Schistosoma mansoni

Share to Schistosomiasis

Source

Identifiers

PMID: 26924460
DOI: 10.1016/j.vph.2016.02.005

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Increased expression of NTPDases 2 and 3 in mesenteric endothelial cells during schistosomiasis favors leukocyte adhesion through P2Y1 receptors.PMID 26924460Read ↗

Extracellular ATP Induces Vascular Inflammation and Atherosclerosis via Purinergic Receptor Y2 in Mice.Semantic proximity Read ↗

Schistosome tegumental ecto-apyrase (SmATPDase1) degrades exogenous pro-inflammatory and pro-thrombotic nucleotides.Semantic proximity Read ↗

Endothelial c-IAP2 Loss Amplifies P2X7 Receptor-Driven Inflammation and Worsens Infection-Associated Pulmonary HypertensionSemantic proximity Read ↗

Overview of the P2 receptors.Semantic proximity Read ↗

Pharmacology and potential physiological significance of schistosome multidrug resistance transportersSemantic proximity Read ↗

The homophilic CD84 receptor is upregulated on platelets in COVID-19 and sepsisSemantic proximity Read ↗

Functional and signaling characterization of the neutrophil FPR2 selective agonist Act-389949Semantic proximity Read ↗

Thrombocytopenia in murine schistosomiasis is associated with platelet uptake by liver macrophages that have a distinct activation phenotypeSemantic proximity Read ↗

At the cross-point of connexins, calcium, and ATP: blocking hemichannels inhibits vasoconstriction of rat small mesenteric arteries.Semantic proximity Read ↗

ATP induces contraction of cultured brain capillary pericytes via activation of P2Y-type purinergic receptors.Semantic proximity Read ↗

THE ACTIN CYTOSKELETON CONTROLS NADPH OXIDASE ACTIVATION AND G PROTEIN RECRUITMENT MEDIATED BY NEUTROPHIL Gαq-COUPLED RECEPTORSSemantic proximity Read ↗

The two formyl peptide receptors differently regulate GPR84-mediated neutrophil NADPH-oxidase activitySemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.