Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Ectopic expression of the striatal-enriched GTPase Rhes elicits cerebellar degeneration and an ataxia phenotype in Huntington's disease

6 Jun 2015

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Genetic Neurodegenerative Diseases
Share to Mitochondrial Function and Pathology
Share to Neurological disorders and treatments

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 26048156
DOI: 10.1016/j.nbd.2015.05.011

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Ectopic expression of the striatal-enriched GTPase Rhes elicits cerebellar degeneration and an ataxia phenotype in Huntington's diseasePMID 26048156Read ↗

Huntington’s Disease is a disorder of the corpus striatum: Focus on Rhes (Ras homologue enriched in the striatum)Semantic proximity Read ↗

Reinstating aberrant mTORC1 activity in Huntington's disease mice improves disease phenotypes.Semantic proximity Read ↗

Rhes suppression enhances disease phenotypes in Huntington's disease mice.Semantic proximity Read ↗

Rhes, a striatal-enriched small G protein, mediates mTOR signaling and L-DOPA-induced dyskinesia.Semantic proximity Read ↗

Striatal Induction and Spread of the Huntington’s Disease Protein: A Novel Rhes RouteSemantic proximity Read ↗

A striatal-enriched intronic GPCR modulates huntingtin levels and toxicity.Semantic proximity Read ↗

Rhes protein transits from neuron to neuron and facilitates mutant huntingtin spreading in the brainSemantic proximity Read ↗

Neuropeptide Y mitigates neuropathology and motor deficits in mouse models of Machado–Joseph diseaseSemantic proximity Read ↗

The GTP-binding protein Rhes modulates dopamine signalling in striatal medium spiny neurons.Semantic proximity Read ↗

Dysfunctional striatal dopamine signaling in Huntington's diseaseSemantic proximity Read ↗

PKA modulates iron trafficking in the striatum via small GTPase, Rhes.Semantic proximity Read ↗

Striatal Overexpression of ΔJunD Resets L-DOPA-Induced Dyskinesia in a Primate Model of Parkinson DiseaseSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.