Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Cessation of contraction induces cardiomyocyte remodeling during zebrafish cardiogenesis.

American journal of physiology. Heart and circulatory physiology - 1 Feb 2014

Yang Jingchun, Hartjes Katherine A, Nelson Timothy J, Xu Xiaolei

Abstract excerpt

Contraction regulates heart development via a complex mechanotransduction process controlled by various mechanical forces. Here, we exploit zebrafish embryos as an in vivo animal model to discern the contribution from different mechanical forces and identify the underlying mechanotransductive signaling pathways of cardiogenesis. We treated 2 days postfertilization zebrafish embryos with Blebbistatin, a myosin II...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Heart
Share to Heterocyclic Compounds, 4 or More Rings

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Mechanotransduction, Cellular

Share to Muscle Development

Share to Myocardial Contraction

Share to Myocardium

Share to Myocytes, Cardiac

Share to Zebrafish Proteins

Source

Open in PubMed Open full text

Identifiers

PMID: 24322613
DOI: 10.1152/ajpheart.00721.2013
PMCID: PMC3920143

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Cessation of contraction induces cardiomyocyte remodeling during zebrafish cardiogenesis.PMID 24322613Read ↗

Zebrafish integrin a3b is required for cardiac contractility and cardiomyocyte proliferation.Semantic proximity Read ↗

Control of cardiac contractions using Cre-lox and degron strategies in zebrafish.Semantic proximity Read ↗

Mef2c factors are required for early but not late addition of cardiomyocytes to the ventricle.Semantic proximity Read ↗

The regenerative capacity of zebrafish reverses cardiac failure caused by genetic cardiomyocyte depletionSemantic proximity Read ↗

Depletion of zebrafish essential and regulatory myosin light chains reduces cardiac function through distinct mechanisms.Semantic proximity Read ↗

A “non-muscle” α-actinin is an intrinsic component of the cardiac Z-disc and regulates sarcomere turnover, contractility, and heart remodelingSemantic proximity Read ↗

Danicamtiv increases myosin recruitment and alters the chemomechanical cross bridge cycle in cardiac muscleSemantic proximity Read ↗

Loss of embryonic neural crest derived cardiomyocytes causes adult onset hypertrophic cardiomyopathy in zebrafishSemantic proximity Read ↗

Regulation of cardiomyocyte behavior in zebrafish trabeculation by Neuregulin 2a signaling.Semantic proximity Read ↗

Cardiac conduction is required to preserve cardiac chamber morphology.Semantic proximity Read ↗

A cardiac myosin light chain kinase regulates sarcomere assembly in the vertebrate heartSemantic proximity Read ↗

Nonmuscle myosin II exerts tension but does not translocate actin in vertebrate cytokinesis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.