Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Role of the Transforming-Growth-Factor-β1 Gene in Late-Onset Alzheimer’s Disease: Implications for the Treatment

1 Feb 2013

Abstract excerpt

Late-onset Alzheimer's disease (LOAD) is the most common form of dementia in the elderly. LOAD has a complex and largely unknown etiology with strong genetic determinants. Genetics of LOAD is known to involve several genetic risk factors among which the Apolipoprotein E (APOE) gene seems to be the major recognized genetic determinant. Recent efforts have been made to identify other genetic factors involved in the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alzheimer's disease research and treatments
Share to Folate and B Vitamins Research
Share to GDF15 and Related Biomarkers

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 24082824
DOI: 10.2174/1389202911314020007

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Role of the Transforming-Growth-Factor-β1 Gene in Late-Onset Alzheimer’s Disease: Implications for the TreatmentPMID 24082824Read ↗

The CC genotype of transforming growth factor-β1 increases the risk of late-onset Alzheimer's disease and is associated with AD-related depression.Semantic proximity Read ↗

Association between polymorphisms in transforming growth factor-β1 and sporadic Alzheimer's disease in a Chinese population.Semantic proximity Read ↗

+10 T/C polymorphisms in the gene of transforming growth factor-beta1 are associated with neurodegeneration and its clinical evolution.Semantic proximity Read ↗

Association study of three polymorphisms of TGF-beta1 gene with Alzheimer's disease.Semantic proximity Read ↗

Dysfunction of TGF-β1 signaling in Alzheimer’s disease: perspectives for neuroprotectionSemantic proximity Read ↗

Analysis of genetic polymorphisms in the transforming growth factor-beta1 gene and the risk of Alzheimer's disease.Semantic proximity Read ↗

Meta-analysis of the transforming growth factor-β1 polymorphisms and susceptibility to Alzheimer's disease.Semantic proximity Read ↗

A TGF-beta1 polymorphism association with dementia and neuropathologies: the HAAS.Semantic proximity Read ↗

Association studies of transforming growth factor-beta 1 and Alzheimer's disease.Semantic proximity Read ↗

TGF-β1 Codon 10 T>C Polymorphism Influences Short-Term Functional and Cognitive Decline in Healthy Oldest-Old Individuals: The Pietà Study.Semantic proximity Read ↗

The Role of BMI1 in Late-Onset Sporadic Alzheimer's Disease.Semantic proximity Read ↗

The gene encoding transforming growth factor beta 1 confers risk of ischemic stroke and vascular dementia.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.