Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Replication and recombination factors contributing to recombination-dependent bypass of DNA lesions by template switch.

PLoS genetics - 11 Nov 2010

Vanoli Fabio, Fumasoni Marco, Szakal Barnabas, Maloisel Laurent, Branzei Dana

Abstract excerpt

Damage tolerance mechanisms mediating damage-bypass and gap-filling are crucial for genome integrity. A major damage tolerance pathway involves recombination and is referred to as template switch. Template switch intermediates were visualized by 2D gel electrophoresis in the proximity of replication forks as X-shaped structures involving sister chromatid junctions. The homologous recombination factor Rad51 is...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Chromosomes, Fungal
Share to DNA-Directed DNA Polymerase

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to DNA, Fungal

Share to DNA Replication

Share to Exonucleases

Share to Genome, Fungal

Share to Replication Protein A

Share to Templates, Genetic

Source

Open in PubMed Open full text

Identifiers

PMID: 21085632
DOI: 10.1371/journal.pgen.1001205
PMCID: PMC2978687

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Replication and recombination factors contributing to recombination-dependent bypass of DNA lesions by template switch.PMID 21085632Read ↗

Rad51 determines pathway usage in post-replication repair.Semantic proximity Read ↗

Access to PCNA by Srs2 and Elg1 Controls the Choice between Alternative Repair Pathways in Saccharomyces cerevisiae.Semantic proximity Read ↗

Polymerase Delta in Eukaryotes: How is It Transiently Exchanged with Specialized DNA Polymerases During Translesion DNA Synthesis?Semantic proximity Read ↗

Slx4 and Fun30/SMARCAD1 coordinate S-phase checkpoint regulation and replication fork protection in response to Top1-DNA crosslinksSemantic proximity Read ↗

Replication intermediate architecture reveals the chronology of DNA damage tolerance pathways at UV-stalled replication forks in human cellsSemantic proximity Read ↗

PARP inhibitor synthetic lethality reveals homologous recombination sub-pathway architectureSemantic proximity Read ↗

Multiple pathways promote short-sequence recombination in Saccharomyces cerevisiae.Semantic proximity Read ↗

Rad9/53BP1 promotes crossover recombination DNA repair by limiting the Sgs1 and Mph1 helicasesSemantic proximity Read ↗

Non-enzymatic roles of human RAD51 at stalled replication forksSemantic proximity Read ↗

DNA2 drives processing and restart of reversed replication forks in human cellsSemantic proximity Read ↗

Rad51 paralog complex Rad55–Rad57 acts as a molecular chaperone during homologous recombinationSemantic proximity Read ↗

Strand invasion by HLTF as a mechanism for template switch in fork rescueSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.