Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Glucocorticoid growth suppression response in 13762NF adenocarcinoma-derived Con8 rat mammary tumor cells is mediated by dominant trans-acting factors.

Cancer research - 15 Nov 1991

Webster M K, Guthrie J, Firestone G L

Abstract excerpt

The in vitro and in vivo growth of Con8 cells, a single cell-derived subclone of the 13762NF-transplantable rat mammary adenocarcinoma, is strongly suppressed by glucocorticoid hormones. Hybrids were formed between glucocorticoid-suppressible Con8.hD6 mammary tumor cells (Con8 transfected with the histidinol dehydrogenase selectable marker) and either glucocorticoid-resistant 8RUV7 mammary tumor cells (derived...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adenocarcinoma
Share to Cell Division
Share to DNA

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Drug Resistance

Share to Hybrid Cells

Share to Phenotype

Share to Trans-Activators

Source

Identifiers

PMID: 1933866

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Glucocorticoid growth suppression response in 13762NF adenocarcinoma-derived Con8 rat mammary tumor cells is mediated by dominant trans-acting factors.PMID 1933866Read ↗

Glucocorticoid-regulated gene in transformed to normal phenotypic reversion.Semantic proximity Read ↗

Evidence for a protein-trafficking gene that rescues the defective glucocorticoid-regulated transport and Golgi retention of mouse mammary tumor virus glycoproteins in a rat hepatoma cell-sorting variant.Semantic proximity Read ↗

Cloning of glucocorticoid-regulated genes in C6/ST1 rat glioma phenotypic reversion.Semantic proximity Read ↗

Activation of the galectin-1 (L-14-I) gene from nonexpressing differentiated cells by fusion with undifferentiated and tumorigenic cells.Semantic proximity Read ↗

Suppression of tumorigenicity of a human lung carcinoma line by nontumorigenic bronchial epithelial cells in somatic cell hybrids.Semantic proximity Read ↗

Glucocorticoid-stimulated CCAAT/enhancer-binding protein alpha expression is required for steroid-induced G1 cell cycle arrest of minimal-deviation rat hepatoma cells.Semantic proximity Read ↗

cis-dominant regulation of CD4 and CD8 gene expression in rat/mouse T cell heterohybridomas.Semantic proximity Read ↗

Estrogen Inducibility of c-Ha-ras Transcription in Breast Cancer CellsSemantic proximity Read ↗

Insulin like growth factor-I independence in rat mammary carcinoma cells: a dominant phenotype in somatic cell hybrid experiments.Semantic proximity Read ↗

The differential capacity of glucocorticoids and progestins to alter chromatin structure and induce gene expression in human breast cancer cells.Semantic proximity Read ↗

Selective incorporation of fibronectin isoforms into the extracellular matrix of hybrids between human fibrosarcoma cells and normal human fibroblasts.Semantic proximity Read ↗

Suppression of cellular transformation by glucocorticoid receptor mutants.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.