Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Glu298Asp eNOS gene polymorphism causes attenuation in nonexercising muscle vasodilatation.

Physiological genomics - 10 Apr 2009

Dias Rodrigo G, Alves Maria-Janieire N N, Pereira Alexandre C, Rondon Maria Urbana P B, Dos Santos Marcelo R, Krieger José E, Krieger Marta H, Negrão Carlos E

Abstract excerpt

The influence of Glu298Asp endothelial nitric oxide synthase (eNOS) polymorphism in exercise-induced reflex muscle vasodilatation is unknown. We hypothesized that nonexercising forearm blood flow (FBF) responses during handgrip isometric exercise would be attenuated in individuals carrying the Asp298 allele. In addition, these responses would be mediated by reduced eNOS function and NO-mediated vasodilatation or...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Blood Pressure
Share to Exercise

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Forearm

Share to Hand Strength

Share to Heart Rate

Share to Isometric Contraction

Share to Nitric Oxide Synthase Type III

Share to Vasodilation

Source

Identifiers

PMID: 19158254
DOI: 10.1152/physiolgenomics.90368.2008

Genes

MBP

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Glu298Asp eNOS gene polymorphism causes attenuation in nonexercising muscle vasodilatation.PMID 19158254Read ↗

The functional consequence of the Glu298Asp polymorphism of the endothelial nitric oxide synthase gene in young healthy volunteers.Semantic proximity Read ↗

Functional gene testing of the Glu298Asp polymorphism of the endothelial NO synthase.Semantic proximity Read ↗

NOS3 Glu298Asp genotype and blood pressure response to endurance training: the HERITAGE family study.Semantic proximity Read ↗

The T-786C and Glu298Asp polymorphisms of the endothelial nitric oxide gene affect the forearm blood flow responses of Caucasian hypertensive patients.Semantic proximity Read ↗

Glu298Asp endothelial nitric oxide synthase gene polymorphism interacts with environmental and dietary factors to influence endothelial function.Semantic proximity Read ↗

In-vivo effects of Glu298Asp endothelial nitric oxide synthase polymorphism.Semantic proximity Read ↗

Endothelial nitric oxide synthase polymorphisms and hypertension.Semantic proximity Read ↗

The Glu298Asp polymorphism of the NOS 3 gene as a determinant of the baseline production of nitric oxide.Semantic proximity Read ↗

Endothelial nitric oxide synthase Glu298Asp gene polymorphism is associated with hypertensive response to exercise in well-controlled hypertensive patients.Semantic proximity Read ↗

Glu298Asp polymorphism influences the beneficial effects of fish oil fatty acids on postprandial vascular function.Semantic proximity Read ↗

Exercise training improves muscle vasodilatation in individuals with T786C polymorphism of endothelial nitric oxide synthase gene.Semantic proximity Read ↗

Relationship between the G894T polymorphism (Glu298Asp variant) in endothelial nitric oxide synthase and nitric oxide-mediated endothelial function in human atherosclerosis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.