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Article

Demonstration of focal p53 expression without genetic alterations in endometriotic lesions.

International journal of gynecological pathology : official journal of the International Society of Gynecological Pathologists - 1 Jul 2001

Nakayama K, Toki T, Zhai Y L, Lu X, Horiuchi A, Nikaido T, Konishi I, Fujii S

Abstract excerpt

Their monoclonal origin (as indicated by recent investigations) indicates the neoplastic nature of most endometriotic lesions. p53, a representative tumor suppressor, regulates cell proliferation, and genetic alterations in p53 are involved in carcinogenesis in a wide variety of human cancers. The aim of this study was to examine endometriotic lesions for p53 expression and genetic alterations in p53. An...

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Identifiers

PMID: 11444197
DOI: 10.1097/00004347-200107000-00004

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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