Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Factor V R506Q mutation-Leiden: an independent risk factor for venous thrombosis but not coronary artery disease.

Journal of thrombosis and thrombolysis - 1 Apr 2001

Irani-Hakime N, Tamim H, Elias G, Choueiry S, Kreidy R, Daccache J L, Almawi W Y

Abstract excerpt

BACKGROUND: A specific point G-A transition at nucleotide position 1691 in the factor V (FV) gene, FV-Leiden, was associated with increased risk of venous thromboembolism (VTE). Insofar as the association of FV-Leiden with coronary artery disease (CAD) remains poorly defined, the aim of this study was to determine the prevalence of FV-Leiden in a sample of 68 VTE patients, 69 CAD patients, and 192 randomly...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Activated Protein C Resistance
Share to Age Distribution

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Coronary Disease

Share to Factor V

Share to Lebanon

Share to Odds Ratio

Share to Thrombophilia

Share to Venous Thrombosis

Source

Identifiers

PMID: 11406725
DOI: 10.1023/a:1011268531377

Genes

CAD

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Factor V R506Q mutation-Leiden: an independent risk factor for venous thrombosis but not coronary artery disease.PMID 11406725Read ↗

Mutation in the gene coding for coagulation factor V and the risk of myocardial infarction, stroke, and venous thrombosis in apparently healthy men.Semantic proximity Read ↗

Seven haemostatic gene polymorphisms in coronary disease: meta-analysis of 66,155 cases and 91,307 controls.Semantic proximity Read ↗

The prevalence of factor V (G1691A), MTHFR (C677T) and PT (G20210A) gene mutations in arterial thrombosis.Semantic proximity Read ↗

The VITA project: prothrombin G20210A mutation and venous thromboembolism in the general population.Semantic proximity Read ↗

Factor V G1691A, prothrombin G20210A, and methylenetetrahydrofolate reductase [MTHFR] C677T gene polymorphism in angiographically documented coronary artery disease.Semantic proximity Read ↗

Association of FV G1691A Polymorphism but not A4070G With Coronary Artery Disease.Semantic proximity Read ↗

Factor V Leiden, prothrombin G20210A, and methylenetetrahydrofolate reductase C677T mutations are not associated with chronic limb ischemia: the Linz Peripheral Arterial Disease (LIPAD) study.Semantic proximity Read ↗

Prevalence of factor V gene mutation amongst myocardial infarction patients and healthy controls is higher in Sweden than in other countries.Semantic proximity Read ↗

Increased prevalence of the G20210A prothrombin gene variant in acute coronary syndromes without metabolic or acquired risk factors or with limited extent of disease.Semantic proximity Read ↗

The prothrombin 20210A allele and the factor V Leiden are associated with venous thrombosis but not with early coronary artery disease.Semantic proximity Read ↗

The factor II G20210A and factor V G1691A gene transitions and coronary heart disease.Semantic proximity Read ↗

G20210A Prothrombin gene variant in Turkish patients with angiographically documented coronary artery disease.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.