Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Increased prevalence of the G20210A prothrombin gene variant in acute coronary syndromes without metabolic or acquired risk factors or with limited extent of disease.

European heart journal - 1 Jan 2002

Burzotta F, Paciaroni K, De Stefano V, Chiusolo P, Manzoli A, Casorelli I, Leone A M, Rossi E, Leone G, Maseri A, Andreotti F

Abstract excerpt

AIMS: To investigate the prevalence of the G20210A prothrombin and G1691A factor V gene variants in patients with acute coronary syndrome stratified according to risk factor profile and to extent of coronary disease, in comparison with matched healthy controls. METHODS AND RESULTS: The 20210 prothrombin and the 1691 factor V loci were genotyped in 247 patients < or =65 years of age (190 myocardial infarction and...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Acute Disease
Share to Coronary Angiography

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Coronary Disease

Share to Factor V

Share to Myocardial Infarction

Share to Prothrombin

Share to Syndrome

Source

Identifiers

PMID: 11741359
DOI: 10.1053/euhj.2001.2685

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Increased prevalence of the G20210A prothrombin gene variant in acute coronary syndromes without metabolic or acquired risk factors or with limited extent of disease.PMID 11741359Read ↗

G20210A prothrombin gene polymorphism and coronary ischaemic syndromes: a phenotype-specific meta-analysis of 12 034 subjects.Semantic proximity Read ↗

G20210A prothrombin gene variant and clinical outcome in patients with a first acute coronary syndrome.Semantic proximity Read ↗

Prevalence of the prothrombin gene variant 20210 G --> A among patients with myocardial infarction.Semantic proximity Read ↗

G20210A prothrombin gene polymorphism and prothrombin activity in subjects with or without angiographically documented coronary artery disease.Semantic proximity Read ↗

Interaction of coagulation defects and cardiovascular risk factors: increased risk of myocardial infarction associated with factor V Leiden or prothrombin 20210A.Semantic proximity Read ↗

Seven haemostatic gene polymorphisms in coronary disease: meta-analysis of 66,155 cases and 91,307 controls.Semantic proximity Read ↗

Combined carrier status of prothrombin 20210A and factor XIII-A Leu34 alleles as a strong risk factor for myocardial infarction: evidence of a gene-gene interaction.Semantic proximity Read ↗

The association of factor V Leiden with myocardial infarction is replicated in 1880 patients with premature disease.Semantic proximity Read ↗

G20210A mutation in prothrombin gene and risk of myocardial infarction, stroke, and venous thrombosis in a large cohort of US men.Semantic proximity Read ↗

G20210A Prothrombin gene variant in Turkish patients with angiographically documented coronary artery disease.Semantic proximity Read ↗

Coagulation Factors II, V, VII, and X, Prothrombin Gene 20210G→A Transition, and Factor V Leiden in Coronary Artery DiseaseSemantic proximity Read ↗

Cardiology patient pages. Prothrombin 20210 mutation (factor II mutation).Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.