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Article

Reversal of β-amyloid induced microglial toxicity <i>in vitro</i> by activation of Fpr2/3

2020-02-14

Abstract excerpt

<h4>Background and Purpose</h4> Microglial inflammatory activity is thought to be a major contributor to the pathology of neurodegenerative conditions such as Alzheimer’s disease (AD), and strategies to restrain their behaviour are under active investigation. Classically, anti-inflammatory approaches aim to suppress pro-inflammatory mediator production, but exploitation of inflammatory resolution, the endogenous...

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Literature Corpus work
f3de0a21-53f1-5667-8e0b-e02fe36a4290
DOI
10.1101/2020.02.13.947051
Open publication

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Reversal of β-amyloid induced microglial toxicity <i>in vitro</i> by activation of Fpr2/3DOI 10.1101/2020.02.13.947051
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