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Article

FPR2 contributes to the dysfunction of inflammation resolution and neuronal excitability in focal cortical dysplasia type IIb and tuberous sclerosis complex

2022-07-21

Abstract excerpt

<title>Abstract</title> <p>Background Focal cortical dysplasia IIb (FCDIIb) and tuberous sclerosis complex (TSC) show the persistent neuroinflammation which promote epileptogenesis and epilepsy progression, suggesting that endogenous resolution of inflammation is inadequate to relieve neuronal network hyperexcitability. To explore the potential roles of formyl peptide receptor 2 (FPR2), which is a key regulator...

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Literature Corpus work
bdc39feb-4773-57cc-8a91-bcb3f188ec8d
DOI
10.21203/rs.3.rs-1843432/v1
Open publication

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FPR2 contributes to the dysfunction of inflammation resolution and neuronal excitability in focal cortical dysplasia type IIb and tuberous sclerosis complexDOI 10.21203/rs.3.rs-1843432/v1
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