Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Neuroinflammation: A Signature or a Cause of Epilepsy?

International journal of molecular sciences - 29 Jun 2021

Pracucci Enrico, Pillai Vinoshene, Lamers Didi, Parra Riccardo, Landi Silvia

Abstract excerpt

Epilepsy can be both a primary pathology and a secondary effect of many neurological conditions. Many papers show that neuroinflammation is a product of epilepsy, and that in pathological conditions characterized by neuroinflammation, there is a higher probability to develop epilepsy. However, the bidirectional mechanism of the reciprocal interaction between epilepsy and neuroinflammation remains to be fully...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Brain Neoplasms
Share to Combined Modality Therapy
Share to Disease Management

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Disease Susceptibility

Share to Epilepsy

Share to Inflammation

Share to Neurodegenerative Diseases

Share to Symptom Assessment

Source

Open in PubMed Open full text

Identifiers

PMID: 34209535
DOI: 10.3390/ijms22136981
PMCID: PMC8267969

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Neuroinflammation: A Signature or a Cause of Epilepsy?PMID 34209535Read ↗

Immunologic and Neuroinflammatory Bases of Epileptogenesis: Personalized Therapeutic OptionsSemantic proximity Read ↗

Neuroinflammation and progressive myoclonus epilepsies: from basic science to therapeutic opportunities.Semantic proximity Read ↗

Reactive Glia-Derived Neuroinflammation: a Novel Hallmark in Lafora Progressive Myoclonus Epilepsy That Progresses with AgeSemantic proximity Read ↗

Targeting Microglial Activation in Drug-Resistant Epilepsy: Emerging Therapeutic StrategiesSemantic proximity Read ↗

Microglia play beneficial roles in multiple experimental seizure modelsSemantic proximity Read ↗

Endocannabinoid signaling in epilepsySemantic proximity Read ↗

Neuropsychiatric symptoms and seizure related with serum cytokine in epilepsy patientsSemantic proximity Read ↗

Diisopropylfluorophosphate-induced status epilepticus drives complex glial cell phenotypes in adult male mice.Semantic proximity Read ↗

Neuroinflammation and Neurodegenerative Diseases: How Much Do We Still Not Know?Semantic proximity Read ↗

Neuroinflammation: Integrated Nervous Tissue Response through Intercellular Interactions at the "Whole System" Scale.Semantic proximity Read ↗

TNF and IL6/Jak2 signaling pathways are the main contributors of the glia-derived neuroinflammation present in Lafora disease, a fatal form of progressive myoclonus epilepsySemantic proximity Read ↗

Innate immune reaction in response to seizures: implications for the neuropathology associated with epilepsy.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.