Article

VSMC phenotype regression induced by hypoxia is the key to unlocking VSMC solid cell cord

2024-02-06

Abstract excerpt

Coronary heart disease is the world's leading cause of death. Vasculogenesis, sprouting angiogenesis, intussusceptive angiogenesis, coalescent angiogenesis, vessel co-option, vasculogenic mimicry and arteriogenesis are the seven main ways of collateral vessel development. However, none of the seven methods of collateral vessel development is sufficient to timely rescue a large number of dying myocardial cells in t...

Topics

Open a Topic to create a Post that cites this publication.

Identifiers and source

Literature Corpus work: 6f4828a3-2e2b-59a7-8ed7-d9396d3d1073
DOI: 10.21203/rs.3.rs-3898893/v1

Open publication

Related research

Semantic proximity does not establish scientific evidence.

Click a neighbor to travelStep 1 · 12 closest

VSMC phenotype regression induced by hypoxia is the key to unlocking VSMC solid cell cordDOI 10.21203/rs.3.rs-3898893/v1Read

Elucidating VSMC phenotypic transition mechanisms to bridge insights into cardiovascular disease implicationsSemantic proximity Read

Contribution of extracellular vesicles to human pulmonary arterial smooth muscle cell dysfunction in congenital heart disease-associated pulmonary arterial hypertensionSemantic proximity Read

A promising field: regulating imbalance of EndMT in cardiovascular diseases.Semantic proximity Read

Research progress on the mechanism of phenotypic transformation of pulmonary artery smooth muscle cells induced by hypoxiaSemantic proximity Read

Mitochondria spatially and temporally modulate VSMC phenotypes via interacting with cytoskeleton in cardiovascular diseases.Semantic proximity Read

Role of Vascular Smooth Muscle Cell Phenotype Switching in Arteriogenesis.Semantic proximity Read

LncRNA MFRL regulates the phenotypic switch of vascular smooth muscle cells to attenuate arterial remodeling by encoding a novel micropeptide MFRLP.Semantic proximity Read

Inhibition of vascular intimal hyperplasia by the myokine Musclin: the role of NPR3/raptor/mTORC1-mediated glycolysis and phenotypic switching of VSMCs.Semantic proximity Read

Oroxylin A Suppresses Pathological Vascular Smooth Muscle Cell Phenotypic Switching and Neointima Formation Through Hindering TSC2/mTORC1/HIF-1-Dependent Glycolysis.Semantic proximity Read

Autophagy promotes the survival and development of tumors by participating in the formation of vasculogenic mimicrySemantic proximity Read

Vascular smooth muscle cell phenotypic transition regulates gap junctions of cardiomyocyte.Semantic proximity Read

Metabolism of vascular smooth muscle cells in vascular diseases.Semantic proximity Read