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Article

Alternatively spliced mdm2 transcripts with loss of p53 binding domain sequences: Transforming ability and frequent detection in human cancer

1 Aug 1996

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 8705862
DOI: 10.1038/nm0896-912

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Related research

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Alternatively spliced mdm2 transcripts with loss of p53 binding domain sequences: Transforming ability and frequent detection in human cancerPMID 8705862Read ↗

Alternative and aberrant splicing of MDM2 mRNA in human cancer.Semantic proximity Read ↗

Expression of alternatively spliced mdm2 transcripts correlates with stabilized wild-type p53 protein in human glioblastoma cellsSemantic proximity Read ↗

Overexpression of MDM2 oncoprotein correlates with possession of estrogen receptor alpha and lack of MDM2 mRNA splice variants in human breast cancerSemantic proximity Read ↗

An Alternative Splice Form of Mdm2 Induces p53-independent Cell Growth and TumorigenesisSemantic proximity Read ↗

Conserved sequence elements in the final exon of MDM2-eight-exon skipping event reveal a ‘cassette regulon’ model of alternative splicing controlled by a distal regulatory elementSemantic proximity Read ↗

MDM4 (MDMX) and its Transcript VariantsSemantic proximity Read ↗

Survivin splice variants and their diagnostic significanceSemantic proximity Read ↗

Alternative and aberrant messenger RNA splicing of the mdm2 oncogene in invasive breast cancer.Semantic proximity Read ↗

Spliced MDM2 isoforms promote mutant p53 accumulation and gain-of-function in tumorigenesis.Semantic proximity Read ↗

A review of mismatch repair gene transcripts: issues for interpretation of mRNA splicing assays.Semantic proximity Read ↗

Mis-splicing Drives Loss of Function of p53 <sup>E224D</sup> Point MutationSemantic proximity Read ↗

Aberrant splicing of the <scp>DMP1</scp>‐<scp>ARF</scp>‐<scp>MDM2</scp>‐p53 pathway in cancerSemantic proximity Read ↗

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