Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The termini of VSV DI particle RNAs are sufficient to signal RNA encapsidation, replication, and budding to generate infectious particles.

Virology - 10 Jan 1995

Pattnaik A K, Ball L A, LeGrone A, Wertz G W

Abstract excerpt

Infectious defective interfering (DI) particles of the negative-stranded RNA virus vesicular stomatitis virus (VSV) have been recovered from negative-sense transcripts of a plasmid that contains a full-length cDNA derived from the DI-T particle genome. In order to determine the cis-acting sequences necessary for RNA replication, encapsidation, and budding and to approximate the minimal size of RNA that can be...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cricetinae
Share to Defective Viruses
Share to Mutation

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to RNA, Viral

Share to Sequence Deletion

Share to Vesicular stomatitis Indiana virus

Share to Virus Replication

Source

Open in PubMed Open full text

Identifiers

PMID: 7831839
DOI: 10.1016/s0042-6822(95)80005-0
PMCID: PMC7131126

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The termini of VSV DI particle RNAs are sufficient to signal RNA encapsidation, replication, and budding to generate infectious particles.PMID 7831839Read ↗

Replication Signals in the Genome of Vesicular Stomatitis Virus and Its Defective Interfering Particles: Identification of a Sequence Element That Enhances DI RNA ReplicationSemantic proximity Read ↗

Overlapping Signals for Transcription and Replication at the 3′ Terminus of the Vesicular Stomatitis Virus GenomeSemantic proximity Read ↗

Replication and amplification of defective interfering particle RNAs of vesicular stomatitis virus in cells expressing viral proteins from vectors containing cloned cDNAsSemantic proximity Read ↗

Regulation of RNA Synthesis by the Genomic Termini of Vesicular Stomatitis Virus: Identification of Distinct Sequences Essential for Transcription but Not ReplicationSemantic proximity Read ↗

The 5'-terminal nucleotides of hepatitis A virus RNA, but not poliovirus RNA, are required for infectivity.Semantic proximity Read ↗

Efficient recovery of infectious vesicular stomatitis virus entirely from cDNA clones.Semantic proximity Read ↗

Mutational analyses of the intergenic dinucleotide and the transcriptional start sequence of vesicular stomatitis virus (VSV) define sequences required for efficient termination and initiation of VSV transcriptsSemantic proximity Read ↗

An authentic 3' noncoding region is necessary for efficient poliovirus replication.Semantic proximity Read ↗

Selection of Functional 5′ cis -Acting Elements Promoting Efficient Sindbis Virus Genome ReplicationSemantic proximity Read ↗

Rescue of a segmented negative-strand RNA virus entirely from cloned complementary DNAsSemantic proximity Read ↗

Exoribonuclease-Resistant RNAs Exist within both Coding and Noncoding Subgenomic RNAsSemantic proximity Read ↗

Functional and genetic plasticities of the poliovirus genome: quasi-infectious RNAs modified in the 5'-untranslated region yield a variety of pseudorevertants.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.