Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Overlapping Signals for Transcription and Replication at the 3′ Terminus of the Vesicular Stomatitis Virus Genome

1 Jan 1999

Abstract excerpt

Transcription and replication signals within the negative-sense genomic RNA of vesicular stomatitis virus (VSV) are located at the 3' terminus. To identify these signals, we have used a transcription- and replication-competent minigenome of VSV to generate a series of deletions spanning the first 47 nucleotides at the 3' terminus of the VSV genome corresponding to the leader gene. Analysis of these mutants for...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Plant Virus Research Studies
Share to Virology and Viral Diseases
Share to Virus-based gene therapy research

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 9847350
DOI: 10.1128/jvi.73.1.444-452.1999

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Overlapping Signals for Transcription and Replication at the 3′ Terminus of the Vesicular Stomatitis Virus GenomePMID 9847350Read ↗

Regulation of RNA Synthesis by the Genomic Termini of Vesicular Stomatitis Virus: Identification of Distinct Sequences Essential for Transcription but Not ReplicationSemantic proximity Read ↗

The termini of VSV DI particle RNAs are sufficient to signal RNA encapsidation, replication, and budding to generate infectious particles.Semantic proximity Read ↗

Replication Signals in the Genome of Vesicular Stomatitis Virus and Its Defective Interfering Particles: Identification of a Sequence Element That Enhances DI RNA ReplicationSemantic proximity Read ↗

Mutational analyses of the intergenic dinucleotide and the transcriptional start sequence of vesicular stomatitis virus (VSV) define sequences required for efficient termination and initiation of VSV transcriptsSemantic proximity Read ↗

Identification of a Minimal Size Requirement for Termination of Vesicular Stomatitis Virus mRNA: Implications for the Mechanism of TranscriptionSemantic proximity Read ↗

Transcript Initiation and 5′-End Modifications Are Separable Events during Vesicular Stomatitis Virus TranscriptionSemantic proximity Read ↗

An authentic 3' noncoding region is necessary for efficient poliovirus replication.Semantic proximity Read ↗

Identification of Internal Sequences in the 3′ Leader Region of Human Respiratory Syncytial Virus That Enhance Transcription and Confer Replication ProcessivitySemantic proximity Read ↗

Initiation of vesicular stomatitis virus mutant polR1 transcription internally at the N gene in vitro.Semantic proximity Read ↗

Transcription and replication initiate at separate sites on the vesicular stomatitis virus genomeSemantic proximity Read ↗

Replication and amplification of defective interfering particle RNAs of vesicular stomatitis virus in cells expressing viral proteins from vectors containing cloned cDNAsSemantic proximity Read ↗

Mutations in the 5' trailer region of a respiratory syncytial virus minigenome which limit RNA replication to one step.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.