Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

PrimPol-mediated repriming elicits gap-filling by template switching and promotes cellular tolerance to cidofovir

14 Nov 2024

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cytomegalovirus and herpesvirus research
Share to DNA Repair Mechanisms
Share to Polyomavirus and related diseases

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 39577201
DOI: 10.1016/j.dnarep.2024.103787

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

PrimPol-mediated repriming elicits gap-filling by template switching and promotes cellular tolerance to cidofovirPMID 39577201Read ↗

Tolerating DNA damage by repriming: Gap filling in the spotlightSemantic proximity Read ↗

Broadening the reach and investigating the potential of prime editors through fully viral gene-deleted adenoviral vector deliverySemantic proximity Read ↗

PRIMPOL ensures robust handoff between on-the-fly and post-replicative DNA lesion bypassSemantic proximity Read ↗

Development of B-to-A base editors by leveraging translesion synthesis polymeraseSemantic proximity Read ↗

Integrase-independent HIV-1 infection is augmented under conditions of DNA damage and produces a viral reservoirSemantic proximity Read ↗

Models for human telomere C-strand fill-in by CST–Polα-primaseSemantic proximity Read ↗

Nucleotide embargo by SAMHD1: A strategy to block retroviral infectionSemantic proximity Read ↗

Translesion Synthesis across Abasic Lesions by Human B-Family and Y-Family DNA Polymerases α, δ, η, ι, κ, and REV1Semantic proximity Read ↗

Recognition of HIV-1 Capsid Licenses Innate Immune Response to Viral InfectionSemantic proximity Read ↗

Host DNA repair factors empower a mechanism of antiviral nucleoside analog resistanceSemantic proximity Read ↗

HIV Vpr modulates the host DNA damage response at two independent steps to damage DNA and repress double-strand DNA break repairSemantic proximity Read ↗

The Integrase: An Overview of a Key Player Enzyme in the Antiviral ScenarioSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.