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Article

The combination of maternal KIR-B and fetal HLA-C2 is associated with decidua basalis acute atherosis in pregnancies with preeclampsia.

Journal of reproductive immunology - 1 Sept 2018

Johnsen Guro M, Størvold Gro L, Drabbels Jos J M, Haasnoot Geert W, Eikmans Michael, Spruyt-Gerritse Marijke J, Alnæs-Katjavivi Patji, Scherjon Sicco A, Redman Christopher W G, Claas Frans H J, Staff Anne Cathrine

Abstract excerpt

Acute atherosis is an arterial lesion most often occurring in pregnancies complicated by preeclampsia, a hypertensive pregnancy disorder. Acute atherosis predominates in the maternal spiral arteries in the decidua basalis layer of the pregnant uterus. This layer forms the fetal-maternal immunological interface, where fetal extravillous trophoblasts interact with maternal immune cells to promote decidual spiral...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to HLA-C Antigens

Share to Immune Tolerance

Share to Plaque, Atherosclerotic

Share to Pre-Eclampsia

Share to Receptors, KIR

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Source

Identifiers

PMID: 30055414
DOI: 10.1016/j.jri.2018.07.005

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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The combination of maternal KIR-B and fetal HLA-C2 is associated with decidua basalis acute atherosis in pregnancies with preeclampsia.PMID 30055414Read ↗

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