Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Suppression of a Natural Killer Cell Response by Simian Immunodeficiency Virus Peptides.

PLoS pathogens - 1 Sept 2015

Schafer Jamie L, Ries Moritz, Guha Natasha, Connole Michelle, Colantonio Arnaud D, Wiertz Emmanuel J, Wilson Nancy A, Kaur Amitinder, Evans David T

Abstract excerpt

Natural killer (NK) cell responses in primates are regulated in part through interactions between two highly polymorphic molecules, the killer-cell immunoglobulin-like receptors (KIRs) on NK cells and their major histocompatibility complex (MHC) class I ligands on target cells. We previously reported that the binding of a common MHC class I molecule in the rhesus macaque, Mamu-A1*002, to the inhibitory receptor...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Coculture Techniques
Share to Epitopes, T-Lymphocyte

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Immune Evasion

Share to Killer Cells, Natural

Share to Macaca mulatta

Share to Receptors, KIR

Share to Simian Acquired Immunodeficiency Syndrome

Share to Simian Immunodeficiency Virus

Source

Open in PubMed Open full text

Identifiers

PMID: 26333068
DOI: 10.1371/journal.ppat.1005145
PMCID: PMC4557930

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Suppression of a Natural Killer Cell Response by Simian Immunodeficiency Virus Peptides.PMID 26333068Read ↗

KIR polymorphisms modulate peptide-dependent binding to an MHC class I ligand with a Bw6 motif.Semantic proximity Read ↗

MHC class I Ligands of Rhesus Macaque Killer-Cell Immunoglobulin-Like ReceptorsSemantic proximity Read ↗

HIV-1 Control by NK Cells via Reduced Interaction between KIR2DL2 and HLA-C∗12:02/C∗14:03.Semantic proximity Read ↗

The killer-cell immunoglobulin-like receptors of macaques.Semantic proximity Read ↗

HLA-Cw*0102-restricted HIV-1 p24 epitope variants can modulate the binding of the inhibitory KIR2DL2 receptor and primary NK cell function.Semantic proximity Read ↗

KIRigami: the case for studying NK cell receptors in SIV+ macaques.Semantic proximity Read ↗

Decreased NK cell frequency and function is associated with increased risk of KIR3DL allele polymorphism in simian immunodeficiency virus-infected rhesus macaques with high viral loads.Semantic proximity Read ↗

Infection with "escaped" virus variants impairs control of simian immunodeficiency virus SIVmac239 replication in Mamu-B*08-positive macaques.Semantic proximity Read ↗

Sequence variations in HIV-1 p24 Gag-derived epitopes can alter binding of KIR2DL2 to HLA-C*03:04 and modulate primary natural killer cell function.Semantic proximity Read ↗

Patterns of CD8+ immunodominance may influence the ability of Mamu-B*08-positive macaques to naturally control simian immunodeficiency virus SIVmac239 replication.Semantic proximity Read ↗

Peptide selectivity discriminates NK cells from KIR2DL2- and KIR2DL3-positive individuals.Semantic proximity Read ↗

NKp30 and NKG2D contribute to natural killer recognition of HIV-infected cellsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.