Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Redox regulation of 14-3-3ζ controls monocyte migration.

Arteriosclerosis, thrombosis, and vascular biology - 1 Jul 2014

Kim Hong Seok, Ullevig Sarah L, Nguyen Huynh Nga, Vanegas Difernando, Asmis Reto

Abstract excerpt

OBJECTIVE: Metabolic stress primes monocytes for accelerated chemokine-mediated adhesion, migration, and recruitment into vascular lesions by increasing actin remodeling. The mechanism linking metabolic stress to accelerated actin turnover and enhanced monocyte migration was not known. We tested the hypothesis that in metabolically primed monocytes, the acceleration of monocyte chemoattractant protein-1-induced...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to 14-3-3 Proteins
Share to Actin Depolymerizing Factors

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Chemokine CCL2

Share to Chemotaxis, Leukocyte

Share to Glutaredoxins

Share to Lim Kinases

Share to Lipoproteins, LDL

Share to Phosphoprotein Phosphatases

Source

Open in PubMed Open full text

Identifiers

PMID: 24812321
DOI: 10.1161/ATVBAHA.114.303746
PMCID: PMC4065841

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 10 closest

Redox regulation of 14-3-3ζ controls monocyte migration.PMID 24812321Read ↗

Rewiring of glucose metabolism defines trained immunity induced by oxidized low-density lipoprotein.Semantic proximity Read ↗

The glycolytic enzyme PKM2 bridges metabolic and inflammatory dysfunction in coronary artery disease.Semantic proximity Read ↗

Monocytic MKP-1 is a Sensor of the Metabolic Environment and Regulates Function and Phenotypic Fate of Monocyte-Derived Macrophages in Atherosclerosis.Semantic proximity Read ↗

Mitogen-activated protein kinase phosphatase 1 (MKP-1) in macrophage biology and cardiovascular disease. A redox-regulated master controller of monocyte function and macrophage phenotype.Semantic proximity Read ↗

Innate immune cell activation and epigenetic remodeling in symptomatic and asymptomatic atherosclerosis in humans in vivo.Semantic proximity Read ↗

A reducing milieu renders cofilin insensitive to phosphatidylinositol 4,5-bisphosphate (PIP2) inhibition.Semantic proximity Read ↗

Glycocalyx Degradation Induces a Proinflammatory Phenotype and Increased Leukocyte Adhesion in Cultured Endothelial Cells under Flow.Semantic proximity Read ↗

Laminar shear stress inhibits endothelial cell metabolism via KLF2-mediated repression of PFKFB3.Semantic proximity Read ↗

Phosphorylation of Ser-3 of cofilin regulates its essential function on actin.Semantic proximity Read ↗

High glucose induces upregulation of scavenger receptors and promotes maturation of dendritic cells.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.