TMEM106B influences volume of left-sided temporal lobe and interhemispheric structures in the general population.

doi:10.1016/j.biopsych.2014.03.006

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

TMEM106B influences volume of left-sided temporal lobe and interhemispheric structures in the general population.

Biological psychiatry - 15 Sept 2014

Adams Hieab H H, Verhaaren Benjamin F J, Vrooman Henri A, Uitterlinden Andre G, Hofman Albert, van Duijn Cornelia M, van der Lugt Aad, Niessen Wiro J, Vernooij Meike W, Ikram M Arfan

Abstract excerpt

BACKGROUND: Frontotemporal lobar degeneration is a neurodegenerative disease characterized by brain atrophy of the frontal and anterior temporal lobes. The associated frontotemporal dementia syndromes are clinically heterogeneous, and the pattern of affected cortical regions varies among subtypes. The TMEM106B rs1990622 polymorphism is associated with frontotemporal lobar degeneration, but little is known about...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Brain

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Frontotemporal Dementia

Share to Functional Laterality

Share to Gray Matter

Share to Magnetic Resonance Imaging

Share to Membrane Proteins

Share to Nerve Tissue Proteins

Share to Temporal Lobe

Source

Open in PubMed

Identifiers

PMID: 24731779
DOI: 10.1016/j.biopsych.2014.03.006

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

TMEM106B influences volume of left-sided temporal lobe and interhemispheric structures in the general population.PMID 24731779Read ↗

Gene-Specific Effects on Brain Volume and Cognition of TMEM106B in Frontotemporal Lobar Degeneration.Semantic proximity Read ↗

Gene specific effects on brain volume and cognition of<i>TMEM106B</i>in frontotemporal lobar degenerationSemantic proximity Read ↗

Genome-wide analysis reveals novel genes influencing temporal lobe structure with relevance to neurodegeneration in Alzheimer's disease.Semantic proximity Read ↗

Disease-modifying effects of TMEM106B in genetic frontotemporal dementia: a longitudinal GENFI studySemantic proximity Read ↗

Effect of Alzheimer's Disease Risk Variant rs3824968 at SORL1 on Regional Gray Matter Volume and Age-Related Interaction in Adult Lifespan.Semantic proximity Read ↗

Effect of TMEM106B polymorphism on functional network connectivity in asymptomatic GRN mutation carriers.Semantic proximity Read ↗

SORL1 rs1699102 polymorphism modulates age-related cognitive decline and gray matter volume reduction in non-demented individuals.Semantic proximity Read ↗

The COMT Val158Met polymorphism and temporal lobe morphometry in healthy adults.Semantic proximity Read ↗

MS4A6A genotypes are associated with the atrophy rates of Alzheimer's disease related brain structures.Semantic proximity Read ↗

Bayesian stroke modeling details sex biases in the white matter substrates of aphasiaSemantic proximity Read ↗

COMT Val158Met × SLC6A4 5-HTTLPR interaction impacts on gray matter volume of regions supporting emotion processing.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.