Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Fibroblast growth factor receptor splice variants are stable markers of oncogenic transforming growth factor β1 signaling in metastatic breast cancers

11 Mar 2014

Abstract excerpt

INTRODUCTION: Epithelial-mesenchymal transition (EMT) and mesenchymal-epithelial transition (MET) facilitate breast cancer (BC) metastasis; however, stable molecular changes that result as a consequence of these processes remain poorly defined. Therefore, with the hope of targeting unique aspects of metastatic tumor outgrowth, we sought to identify molecular markers that could identify tumor cells that had...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Angiogenesis and VEGF in Cancer
Share to Cancer Cells and Metastasis
Share to Fibroblast Growth Factor Research

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 24618085
DOI: 10.1186/bcr3623

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Fibroblast growth factor receptor splice variants are stable markers of oncogenic transforming growth factor β1 signaling in metastatic breast cancersPMID 24618085Read ↗

Maintenance of epithelial traits and resistance to mesenchymal reprogramming promote proliferation in metastatic breast cancerSemantic proximity Read ↗

Exon-level transcriptome profiling in murine breast cancer reveals splicing changes specific to tumors with different metastatic abilities.Semantic proximity Read ↗

FGFR4 regulates tumor subtype differentiation in luminal breast cancer and metastatic diseaseSemantic proximity Read ↗

Integrins Alpha-2 and Beta-1 expression increases through Multiple Generations of the EDW01 Patient-Derived Xenograft Model of Breast Cancer - insight into their role in Epithelial Mesenchymal Transition in vivo gained from an in vitro model systemSemantic proximity Read ↗

Resistance to mesenchymal reprogramming sustains clonal propagation in metastatic breast cancerSemantic proximity Read ↗

Multiple receptor tyrosine kinases promote the in vitro phenotype of metastatic human osteosarcoma cell linesSemantic proximity Read ↗

FGFR2 controls growth, adhesion and migration of nontumorigenic human mammary epithelial cells by regulation of integrin β1 degradationSemantic proximity Read ↗

CD44 splice isoform switching in human and mouse epithelium is essential for epithelial-mesenchymal transition and breast cancer progressionSemantic proximity Read ↗

Metastasis-associated wound repair promotes reciprocal activation of the lung epithelium and breast cancer metastases during outgrowthSemantic proximity Read ↗

Role of c-Met/β1 integrin complex in the metastatic cascadeSemantic proximity Read ↗

Regulation of breast cancer metastasis by Runx2 and estrogen signaling: the role of SNAI2Semantic proximity Read ↗

Expression of the FGFR2 mesenchymal splicing variant in epithelial cells drives epithelial-mesenchymal transitionSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.