Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

T-type Ca2+ channels in absence epilepsy.

Pflugers Archiv : European journal of physiology - 1 Apr 2014

Cheong Eunji, Shin Hee-Sup

Abstract excerpt

Absence epilepsy accompanies the paroxysmal oscillations in the thalamocortical circuit referred as spike and wave discharges (SWDs). Low-threshold burst firing mediated by T-type Ca(2+) channels highly expressed in both inhibitory thalamic reticular nuclei (TRN) and excitatory thalamocortical (TC) neurons has been correlated with the generation of SWDs. A generally accepted view has been that rhythmic burst...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Action Potentials
Share to Calcium Channels, T-Type
Share to Electroencephalography

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Epilepsy, Absence

Share to Mutation

Share to Thalamus

Source

Identifiers

PMID: 24519464
DOI: 10.1007/s00424-014-1461-y

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

T-type Ca2+ channels in absence epilepsy.PMID 24519464Read ↗

Genetic studies on the role of T-type Ca2+ channels in sleep and absence epilepsy.Semantic proximity Read ↗

T-type Ca2+ channels and absence epilepsy.Semantic proximity Read ↗

Role of the alpha1G T-type calcium channel in spontaneous absence seizures in mutant mice.Semantic proximity Read ↗

Modeling pathogenesis and treatment response in childhood absence epilepsySemantic proximity Read ↗

Actions of the Anti-Seizure Drug Carbamazepine in the Thalamic Reticular Nucleus: Potential Mechanism of Aggravating Absence SeizuresSemantic proximity Read ↗

The role of calcium channel mutations in human epilepsy.Semantic proximity Read ↗

Spike-and-wave discharges of absence seizures in a sleep waves-constrained corticothalamic modelSemantic proximity Read ↗

The role of T-type calcium channels in epilepsy and pain.Semantic proximity Read ↗

Functional characterization and neuronal modeling of the effects of childhood absence epilepsy variants of CACNA1H, a T-type calcium channel.Semantic proximity Read ↗

Higher-order thalamic nuclei facilitate the generalization and maintenance of spike-and-wave discharges of absence seizuresSemantic proximity Read ↗

CaV3.2 calcium channels control NMDA receptor-mediated transmission: a new mechanism for absence epilepsy.Semantic proximity Read ↗

Isolated P/Q Calcium Channel Deletion in Layer VI Corticothalamic Neurons Generates Absence Epilepsy.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.