Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Calcium-induced outgrowth of astrocytic peripheral processes requires actin binding by Profilin-1.

Cell calcium - 1 Jan 2000

Molotkov Dmitry, Zobova Svetlana, Arcas Jose Miguel, Khiroug Leonard

Abstract excerpt

Peripheral astrocytic processes (PAPs) are highly motile structures that are strategically positioned in close proximity to synapses. Long-lasting PAP retraction in hypothalamus is known to alter synaptic transmission. The PAP motility is likely to be actin-based because they are known to contain actin-related proteins such as Ezrin. However, the link between dynamic activity-dependent changes in astrocytic...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Astrocytes
Share to Calcium Signaling
Share to Cell Growth Processes

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Cell Surface Extensions

Share to Genetic Engineering

Share to Photolysis

Share to Profilins

Share to Synaptic Transmission

Source

Identifiers

PMID: 23578580
DOI: 10.1016/j.ceca.2013.03.001

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Calcium-induced outgrowth of astrocytic peripheral processes requires actin binding by Profilin-1.PMID 23578580Read ↗

Ezrin deficiency triggers glial fibrillary acidic protein upregulation and a distinct reactive astrocyte phenotype.Semantic proximity Read ↗

The Alexander Disease Protein GFAP Drives Mitochondrial FissionSemantic proximity Read ↗

“Generation of morphologically distinct astrocyte subtypes from human iPSCs reveals a link between glial shape, function and reactivity”Semantic proximity Read ↗

Regulation of neurite growth by spontaneous Ca2+ oscillations in astrocytes.Semantic proximity Read ↗

A phosphatidylinositol lipids system, lamellipodin, and Ena/VASP regulate dynamic morphology of multipolar migrating cells in the developing cerebral cortex.Semantic proximity Read ↗

Dynamics of mutated GFAP aggregates revealed by real-time imaging of an astrocyte model of Alexander disease.Semantic proximity Read ↗

Reversible, activity-dependent targeting of profilin to neuronal nuclei.Semantic proximity Read ↗

Plasticity in astrocytic phenotypes. A role for protein kinase C, tyrosine kinases, and cytoskeleton signaling.Semantic proximity Read ↗

Longest neurite-specific activation of Rap1B in hippocampal neurons contributes to polarity formation through RalA and Nore1A in addition to PI3-kinase.Semantic proximity Read ↗

Perspectives for Ezrin and Radixin in Astrocytes: Kinases, Functions and PathologySemantic proximity Read ↗

The transition of microglia to a ramified phenotype is associated with the formation of stable acetylated and detyrosinated microtubules.Semantic proximity Read ↗

Profilin1 is required for glial cell adhesion and radial migration of cerebellar granule neurons.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.