Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Long-term bisphenol A exposure accelerates insulitis development in diabetes-prone NOD mice.

Immunopharmacology and immunotoxicology - 1 Jun 2013

Bodin Johanna, Bølling Anette Kocbach, Samuelsen Mari, Becher Rune, Løvik Martinus, Nygaard Unni Cecilie

Abstract excerpt

Exposure to the endocrine disruptor (ED) bisphenol A (BPA) used in polycarbonate plastic and epoxy resins appears ubiquitous since BPA can be found in over 90% of analyzed urine samples from all age groups. There is a parallel occurrence of increased prevalence in type 1 diabetes mellitus (T1DM) and an increased exposure to EDs the last decades. T1DM is caused by insulin deficiency due to autoimmune destruction...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Benzhydryl Compounds
Share to Diabetes Mellitus, Experimental

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Endocrine Disruptors

Share to Insulin-Secreting Cells

Share to Insulin Secretion

Share to Mice, Inbred NOD

Share to Pancreas

Share to Phenols

Source

Identifiers

PMID: 23496298
DOI: 10.3109/08923973.2013.772195

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Long-term bisphenol A exposure accelerates insulitis development in diabetes-prone NOD mice.PMID 23496298Read ↗

Can Exposure to Environmental Chemicals Increase the Risk of Diabetes Type 1 Development?Semantic proximity Read ↗

Exposure To Persistent Organic Pollutants Alters The Serum Metabolome In Non-Obese Diabetic MiceSemantic proximity Read ↗

Gut dysbiosis and impairment of immune system homeostasis in perinatally-exposed mice to Bisphenol A precede obese phenotype developmentSemantic proximity Read ↗

Long-term in vivo polychlorinated biphenyl 126 exposure induces oxidative stress and alters proteomic profile on islets of LangerhansSemantic proximity Read ↗

Prolonged low-dose dioxin exposure impairs metabolic adaptability to high-fat diet feeding in female but not male miceSemantic proximity Read ↗

Dysregulated B7-1 and B7-2 expression on nonobese diabetic mouse B cells is associated with increased T cell costimulation and the development of insulitis.Semantic proximity Read ↗

Perinatal bisphenol A exposure and adult glucose homeostasis: identifying critical windows of exposure.Semantic proximity Read ↗

2α-Methyl-19-nor-(20S)-1,25-dihydroxyvitamin D(3) protects the insulin 2 knockout non-obese diabetic mouse from developing type 1 diabetes without hypercalcaemia.Semantic proximity Read ↗

Bisphenol A disruption promotes mammary tumor microenvironment via phenotypic cell polarization and inflammatory response.Semantic proximity Read ↗

Single and combined effect of bisphenol A with high sucrose diet on the diabetic and renal tubular dysfunction phenotypes in Drosophila melanogaster.Semantic proximity Read ↗

E2F1-deficient NOD/SCID mice are an experimental model for dry mouth.Semantic proximity Read ↗

Non-alcoholic Fatty Liver Disease Induced by Perinatal Exposure to Bisphenol a Is Associated With Activated mTOR and TLR4/NF-κB Signaling Pathways in Offspring RatsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.