Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Perinatal bisphenol A exposure and adult glucose homeostasis: identifying critical windows of exposure.

PloS one - 1 Jan 2013

Liu Jingli, Yu Pan, Qian Wenyi, Li Yan, Zhao Jingjing, Huan Fei, Wang Jun, Xiao Hang

Abstract excerpt

Bisphenol A (BPA) is a widespread endocrine-disrupting chemical used as the building block for polycarbonate plastics. Epidemiological evidence has correlated BPA exposure with higher risk of heart disease and type 2 diabetes. However, it remains unknown whether there are critical windows of susceptibility to BPA exposure on the development of dysglycemia. This study was an attempt to investigate the critical...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Benzhydryl Compounds
Share to Caspase 3
Share to Cyclin D1

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Insulin-Secreting Cells

Share to Islets of Langerhans

Share to Phenols

Share to Pregnancy Outcome

Share to Prenatal Exposure Delayed Effects

Source

Open in PubMed Open full text

Identifiers

PMID: 23675523
DOI: 10.1371/journal.pone.0064143
PMCID: PMC3651242

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Perinatal bisphenol A exposure and adult glucose homeostasis: identifying critical windows of exposure.PMID 23675523Read ↗

Gut dysbiosis and impairment of immune system homeostasis in perinatally-exposed mice to Bisphenol A precede obese phenotype developmentSemantic proximity Read ↗

Bisphenol A Exposure Disrupts Metabolic Health Across Multiple Generations in the MouseSemantic proximity Read ↗

Perinatal exposure to bisphenol A exacerbates nonalcoholic steatohepatitis-like phenotype in male rat offspring fed on a high-fat diet.Semantic proximity Read ↗

Liver DNA methylation analysis in adult female C57BL/6JxFVB mice following perinatal exposure to bisphenol A.Semantic proximity Read ↗

Environmental cadmium exposure during pregnancy causes diabetes-like phenotypes in mouse offspring: Association with oxidative stress in the fetal liver.Semantic proximity Read ↗

Prenatal exposure to bisphenol A disrupts mouse fetal lung developmentSemantic proximity Read ↗

Epigenetic perspective on the developmental effects of bisphenol A.Semantic proximity Read ↗

Consequences of a compromised intrauterine environment on islet functionSemantic proximity Read ↗

Perinatal lead (Pb) exposure results in sex-specific effects on food intake, fat, weight, and insulin response across the murine life-course.Semantic proximity Read ↗

Does Bisphenol A Confer Risk of Neurodevelopmental Disorders? What We’ve Learned from Developmental Neurotoxicity Studies in Animal ModelsSemantic proximity Read ↗

Long-term bisphenol A exposure accelerates insulitis development in diabetes-prone NOD mice.Semantic proximity Read ↗

Bisphenol a exposure disrupts genomic imprinting in the mouse.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.