Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Non-alcoholic Fatty Liver Disease Induced by Perinatal Exposure to Bisphenol a Is Associated With Activated mTOR and TLR4/NF-κB Signaling Pathways in Offspring Rats

10 Sept 2019

Abstract excerpt

Accumulating evidence suggests a role of bisphenol A (BPA) in non-alcoholic fatty liver disease (NAFLD), and its mechanism may be related to the up-regulation of lipogenic genes, but the mechanism of BPA induced lipogenic gene expression remains unknown. The aim of this study was to investigate the effects of perinatal exposure to BPA on NAFLD and its mechanisms. Pregnant Sprague-Dawley rats had access to...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Birth, Development, and Health
Share to Effects and risks of endocrine disrupting chemicals
Share to Fatty Acid Research and Health

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 31551937
DOI: 10.3389/fendo.2019.00620

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Non-alcoholic Fatty Liver Disease Induced by Perinatal Exposure to Bisphenol a Is Associated With Activated mTOR and TLR4/NF-κB Signaling Pathways in Offspring RatsPMID 31551937Read ↗

Perinatal exposure to bisphenol A exacerbates nonalcoholic steatohepatitis-like phenotype in male rat offspring fed on a high-fat diet.Semantic proximity Read ↗

Potentiation of Adipogenesis by Reactive Oxygen Species is a Unifying Mechanism in the Pro-adipogenic Properties of Bisphenol A and its New Structural AnaloguesSemantic proximity Read ↗

Sex-specific expression of the human NAFLD-NASH transcriptional signatures in the liver of medaka with a history of ancestral bisphenol A exposureSemantic proximity Read ↗

Sex, Nutrition, and NAFLD: Relevance of Environmental PollutionSemantic proximity Read ↗

The PCOS-NAFLD Multidisease Phenotype Occurred in Medaka Fish Four Generations after the Removal of Bisphenol A Exposure.Semantic proximity Read ↗

Medaka liver developed Human NAFLD-NASH transcriptional signatures in response to ancestral bisphenol A exposureSemantic proximity Read ↗

The estrogenic endocrine disrupting chemical bisphenol A (BPA) and obesity.Semantic proximity Read ↗

Prenatal Bisphenol A Exposure in Mice Induces Multi-tissue Multi-omics Disruptions Linking to Cardiometabolic DisordersSemantic proximity Read ↗

Prenatal ethanol exposure programs an increased susceptibility of non-alcoholic fatty liver disease in female adult offspring rats.Semantic proximity Read ↗

Curcumin protects against bisphenol A -induced hepatic steatosis by modulating gut microbiota and liver fat synthesis in CD-1 miceSemantic proximity Read ↗

Developmental Programming of NAFLD by Parental ObesitySemantic proximity Read ↗

Oxidative stress and altered lipid homeostasis in the programming of offspring fatty liver by maternal obesity.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.