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Article

ICAM1 K469E and E-selectin S128R polymorphisms could predispose to increased autoantibody production and TSH suppression in Graves' disease.

Molecular biology reports - 1 Mar 2013

Kanmaz-Özer Müge, Doğru-Abbasoğlu Semra, Vural Pervin, Özderya Ayşenur, Karadağ Berrin, Uysal Müjdat

Abstract excerpt

The etiopathogenesis of Graves' disease (GD) has not been clearly elucidated although the role of chronical inflammation and endothelial dysfunction has been established. Adhesion molecules such as intercellular adhesion molecule 1 (ICAM1), vascular cell adhesion molecule 1 (VCAM1), and E-selectin are secreted from vascular endothelium and promote accumulation of leukocytes in damaged endothelial areas. This...

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Source

Identifiers

PMID: 23242661
DOI: 10.1007/s11033-012-2359-4

Genes

ICAM1
VCAM1
TPO

Variants

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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