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Article

APOBEC3G expression and hypermutation are inversely associated with human immunodeficiency virus type 1 (HIV-1) burden in vivo

12 May 2012

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 22579353
DOI: 10.1016/j.virol.2012.03.018

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Related research

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Click a neighbor to travelStep 1 · 12 closest

APOBEC3G expression and hypermutation are inversely associated with human immunodeficiency virus type 1 (HIV-1) burden in vivoPMID 22579353Read ↗

APOBEC3F Properties and Hypermutation Preferences Indicate Activity against HIV-1 In VivoSemantic proximity Read ↗

Antiretroviral APOBEC3 Cytidine Deaminases Alter HIV-1 Provirus Integration Site ProfilesSemantic proximity Read ↗

Human APOBEC3 Variations and Viral InfectionSemantic proximity Read ↗

Extremely High Mutation Rate of HIV-1 In VivoSemantic proximity Read ↗

In vivo HIV-1 hypermutation and viral loads among antiretroviral-naive Brazilian patients.Semantic proximity Read ↗

Interactions of host APOBEC3 restriction factors with HIV-1 in vivo: implications for therapeuticsSemantic proximity Read ↗

Leveraging APOBEC3 Proteins to Alter the HIV Mutation Rate and Combat AIDSSemantic proximity Read ↗

APOBEC3B deletion and risk of HIV-1 acquisition.Semantic proximity Read ↗

Human immunodeficiency virus (HIV) type 1 proviral hypermutation correlates with CD4 count in HIV-infected women from Kenya.Semantic proximity Read ↗

The Disassociation of A3G-Related HIV-1 cDNA G-to-A Hypermutation to Viral Infectivity.Semantic proximity Read ↗

Antiretroviral APOBEC3 cytidine deaminases alter HIV-1 provirus integration site profilesSemantic proximity Read ↗

APOBEC3G and APOBEC3F rarely co-mutate the same HIV genome.Semantic proximity Read ↗

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