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Article

CRISPR Immunity Relies on the Consecutive Binding and Degradation of Negatively Supercoiled Invader DNA by Cascade and Cas3

19 Apr 2012

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 22521689
DOI: 10.1016/j.molcel.2012.03.018

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Related research

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Research trail→

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CRISPR Immunity Relies on the Consecutive Binding and Degradation of Negatively Supercoiled Invader DNA by Cascade and Cas3PMID 22521689Read ↗

CRISPR-Cas Systems: Prokaryotes Upgrade to Adaptive ImmunitySemantic proximity Read ↗

A CRISPR-associated factor Csa3a regulates DNA damage repair in Crenarchaeon Sulfolobus islandicus.Semantic proximity Read ↗

Structural basis of Cas3 activation in type I-C CRISPR-Cas systemSemantic proximity Read ↗

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Cas9 interaction with the tracrRNA nexus modulates the repression of type II-A CRISPR-cas genes.Semantic proximity Read ↗

Conformational Control of Cascade Interference and Priming Activities in CRISPR Immunity.Semantic proximity Read ↗

A CRISPR evolutionary arms race: structural insights into viral anti-CRISPR/Cas responsesSemantic proximity Read ↗

Two distinct DNA binding modes guide dual roles of a CRISPR-Cas protein complex.Semantic proximity Read ↗

Direct observation of R-loop formation by single RNA-guided Cas9 and Cascade effector complexes.Semantic proximity Read ↗

CRISPR-Cas12a targeting of ssDNA plays no detectable role in immunitySemantic proximity Read ↗

A target expression threshold dictates invader defense and autoimmunity by CRISPR-Cas13Semantic proximity Read ↗

Adaptation in bacterial CRISPR-Cas immunity can be driven by defective phages.Semantic proximity Read ↗

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