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Article

Long-term Fgf23 deficiency does not influence aging, glucose homeostasis, or fat metabolism in mice with a nonfunctioning vitamin D receptor.

Endocrinology - 1 Apr 2012

Streicher Carmen, Zeitz Ute, Andrukhova Olena, Rupprecht Anne, Pohl Elena, Larsson Tobias E, Windisch Wilhelm, Lanske Beate, Erben Reinhold G

Abstract excerpt

It is still controversial whether the bone-derived hormone fibroblast growth factor-23 (FGF23) has additional physiological functions apart from its well-known suppressive actions on renal phosphate reabsorption and vitamin D hormone synthesis. Here we analyzed premature aging, mineral homeostasi...

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Source

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Identifiers

PMID: 22294750
DOI: 10.1210/en.2011-1878
PMCID: PMC3320267

Genes

VDR
FGF23

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-22294750-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

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Long-term Fgf23 deficiency does not influence aging, glucose homeostasis, or fat metabolism in mice with a nonfunctioning vitamin D receptor.PMID 22294750Read ↗

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