Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Cutting edge: an NK cell-independent role for Slamf4 in controlling humoral autoimmunity.

Journal of immunology (Baltimore, Md. : 1950) - 1 Jul 2011

Brown Daniel R, Calpe Silvia, Keszei Marton, Wang Ninghai, McArdel Shannon, Terhorst Cox, Sharpe Arlene H

Abstract excerpt

Several genes within a syntenic region of human and mouse chromosome 1 are associated with predisposition to systemic lupus erythematosus. Analyses of lupus-prone congenic mice have pointed to an important role for the signaling lymphocyte activation molecule family (slamf)6 surface receptor in l...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Autoantibodies
Share to Chromatin
Share to Chronic Disease

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Graft vs Host Disease

Share to Immune Tolerance

Share to Killer Cells, Natural

Share to Lupus Erythematosus, Systemic

Share to Receptors, Immunologic

Source

Open in PubMed Open full text

Identifiers

PMID: 21622868
DOI: 10.4049/jimmunol.1100510
PMCID: PMC3119727

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-21622868-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Cutting edge: an NK cell-independent role for Slamf4 in controlling humoral autoimmunity.PMID 21622868Read ↗

CRISPR-Mediated Triple Knockout of SLAMF1, SLAMF5 and SLAMF6 Supports Positive Signaling Roles in NKT Cell DevelopmentSemantic proximity Read ↗

Association of extensive polymorphisms in the SLAM/CD2 gene cluster with murine lupus.Semantic proximity Read ↗

SLAM family receptors and the SLAM-associated protein (SAP) modulate T cell functions.Semantic proximity Read ↗

Development of murine lupus involves the combined genetic contribution of the SLAM and FcgammaR intervals within the Nba2 autoimmune susceptibility locus.Semantic proximity Read ↗

B cell OX40L supports T follicular helper cell development and contributes to SLE pathogenesisSemantic proximity Read ↗

Identification of IRAK1 as a risk gene with critical role in the pathogenesis of systemic lupus erythematosus.Semantic proximity Read ↗

SH2D1A regulates T-dependent humoral autoimmunity.Semantic proximity Read ↗

Engagement of SLAMF3 enhances CD4 <sup>+</sup> T-cell sensitivity to IL-2 and favors regulatory T-cell polarization in systemic lupus erythematosusSemantic proximity Read ↗

Rare <i>SH2B3</i> coding variants identified in lupus patients impair B cell tolerance and predispose to autoimmunitySemantic proximity Read ↗

Genetic dissection of systemic lupus erythematosus pathogenesis: Sle2 on murine chromosome 4 leads to B cell hyperactivity.Semantic proximity Read ↗

Regulation of Cellular and Humoral Immune Responses by the SLAM and SAP Families of MoleculesSemantic proximity Read ↗

SAP regulation of follicular helper CD4 T cell development and humoral immunity is independent of SLAM and Fyn kinase.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.