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Article

Generation of antibodies of distinct subclasses and specificity is linked to H2s in an active mouse model of epidermolysis bullosa acquisita.

The Journal of investigative dermatology - 1 Jan 2011

Ludwig Ralf J, Recke Andreas, Bieber Katja, Müller Susen, Marques Andreia de Castro, Banczyk David, Hirose Misa, Kasperkiewicz Michael, Ishii Norito, Schmidt Enno, Westermann Jürgen, Zillikens Detlef, Ibrahim Saleh M

Abstract excerpt

Epidermolysis bullosa acquisita (EBA) is an autoimmune blistering disease, characterized by antibodies to type VII collagen (COL7). EBA can be induced in mice by immunization with a fragment of the non-collagenous 1 domain of murine COL7. Contrary to other autoimmune diseases, e.g., rheumatoid arthritis, little is known about the genetic susceptibility for EBA. We therefore used the EBA mouse model to address the...

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PMID: 20720563
DOI: 10.1038/jid.2010.248

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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Generation of antibodies of distinct subclasses and specificity is linked to H2s in an active mouse model of epidermolysis bullosa acquisita.PMID 20720563Read ↗

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