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Article

Autoantibodies to Multiple Epitopes on the Non-Collagenous-1 Domain of Type VII Collagen Induce Blisters.

The Journal of investigative dermatology - 1 Jun 2015

Vorobyev Artem, Ujiie Hideyuki, Recke Andreas, Buijsrogge Jacqueline J A, Jonkman Marcel F, Pas Hendri H, Iwata Hiroaki, Hashimoto Takashi, Kim Soo-Chan, Hoon Kim Jong, Groves Richard, Samavedam Unni, Gupta Yask, Schmidt Enno, Zillikens Detlef, Shimizu Hiroshi, Ludwig Ralf J

Abstract excerpt

Epidermolysis bullosa acquisita (EBA) is an autoimmune blistering disease of the skin and mucous membranes, characterized by autoantibodies against type VII collagen (COL7), a major component of anchoring fibrils. Different clinical EBA phenotypes are described, including mechanobullous and inflammatory variants. Most EBA patients' sera react with epitopes located within the non-collagenous 1 (NC1) domain of...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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PMID: 25689103
DOI: 10.1038/jid.2015.51

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

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Autoantibodies to Multiple Epitopes on the Non-Collagenous-1 Domain of Type VII Collagen Induce Blisters.PMID 25689103Read ↗

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